about
Loss-of-Function Mutation in APC2 Causes Sotos Syndrome FeaturesSPIG1 negatively regulates BDNF maturationProtein tyrosine phosphatase receptor type Z is involved in hippocampus-dependent memory formation through dephosphorylation at Y1105 on p190 RhoGAPProtein tyrosine phosphatase receptor type Z is inactivated by ligand-induced oligomerizationSAP97 promotes the stability of Nax channels at the plasma membraneTyrosine phosphorylation of ErbB4 is enhanced by PSD95 and repressed by protein tyrosine phosphatase receptor type ZSpecific dephosphorylation at tyr-554 of git1 by ptprz promotes its association with paxillin and hic-5.Channel properties of Nax expressed in neurons.Small-molecule inhibition of PTPRZ reduces tumor growth in a rat model of glioblastoma.Protein tyrosine phosphatase receptor type z negatively regulates oligodendrocyte differentiation and myelination.Immunochemical characterization of recombinant and native tropomyosins as a new allergen from the house dust mite, Dermatophagoides farinae.Helicobacter pylori VacA activates the p38/activating transcription factor 2-mediated signal pathway in AZ-521 cells.Plasmin-mediated processing of protein tyrosine phosphatase receptor type Z in the mouse brain.Helicobacter pylori CagA containing ITAM-like sequences localized to lipid rafts negatively regulates VacA-induced signaling in vivo.Mice deficient in protein tyrosine phosphatase receptor type Z are resistant to gastric ulcer induction by VacA of Helicobacter pylori.Directional neuronal migration is impaired in mice lacking adenomatous polyposis coli 2.Age-dependent enhancement of hippocampal long-term potentiation and impairment of spatial learning through the Rho-associated kinase pathway in protein tyrosine phosphatase receptor type Z-deficient mice.Genetic deletion of vesicular monoamine transporter-2 (VMAT2) reduces dopamine transporter activity in mesencephalic neurons in primary culture.Inactivation of Protein Tyrosine Phosphatase Receptor Type Z by Pleiotrophin Promotes Remyelination through Activation of Differentiation of Oligodendrocyte Precursor Cells.Autoimmunity to the sodium-level sensor in the brain causes essential hypernatremia.
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Akihiro Fujikawa
@ast
Akihiro Fujikawa
@en
Akihiro Fujikawa
@es
Akihiro Fujikawa
@nl
Akihiro Fujikawa
@sl
type
label
Akihiro Fujikawa
@ast
Akihiro Fujikawa
@en
Akihiro Fujikawa
@es
Akihiro Fujikawa
@nl
Akihiro Fujikawa
@sl
prefLabel
Akihiro Fujikawa
@ast
Akihiro Fujikawa
@en
Akihiro Fujikawa
@es
Akihiro Fujikawa
@nl
Akihiro Fujikawa
@sl
P106
P108
P21
P31
P496
0000-0002-3950-0550