about
A low dose of fermented soy germ alleviates gut barrier injury, hyperalgesia and faecal protease activity in a rat model of inflammatory bowel diseaseCytokine-mediated control of lipopolysaccharide-induced activation of small intestinal epithelial cells.Secretory IgA mediates retrotranscytosis of intact gliadin peptides via the transferrin receptor in celiac disease.Maternally acquired genotoxic Escherichia coli alters offspring's intestinal homeostasis.Perinatal exposure to a low dose of bisphenol A impaired systemic cellular immune response and predisposes young rats to intestinal parasitic infection.Specific IgG response against Mycobacterium avium paratuberculosis in children and adults with Crohn's disease.Prenatal intestinal obstruction affects the myenteric plexus and causes functional bowel impairment in fetal rat experimental model of intestinal atresiaOral tolerance failure upon neonatal gut colonization with Escherichia coli producing the genotoxin colibactinInnate immune recognition on the intestinal mucosa.Consequences of bisphenol a perinatal exposure on immune responses and gut barrier function in mice.Effects of specific lactic acid bacteria on the intestinal permeability to macromolecules and the inflammatory condition.In vitro and ex vivo activation of the TLR5 signaling pathway in intestinal epithelial cells by a commensal Escherichia coli strain.Interactions among secretory immunoglobulin A, CD71, and transglutaminase-2 affect permeability of intestinal epithelial cells to gliadin peptides.Paneth Cell Defects Induce Microbiota Dysbiosis in Mice and Promote Visceral Hypersensitivity.Gut dysbiosis and impairment of immune system homeostasis in perinatally-exposed mice to Bisphenol A precede obese phenotype development.Food intolerance at adulthood after perinatal exposure to the endocrine disruptor bisphenol A.Interleukin-13-mediated paneth cell degranulation and antimicrobial peptide release.Early life stress in mice is a suitable model for Irritable Bowel Syndrome but does not predispose to colitis nor increase susceptibility to enteric infectionsParacellular versus Transcellular Intestinal Permeability to Gliadin Peptides in Active Celiac DiseaseStimulation of Immunity Without Alteration of Oral Tolerance in Mice Fed With Heat-Treated Fermented Infant FormulaEffect of rebamipide on the colonic barrier in interleukin-10-deficient micePathways of gliadin transport in celiac diseaseAuthor Correction: Dimorphic metabolic and endocrine disorders in mice lacking the constitutive androstane receptorDimorphic metabolic and endocrine disorders in mice lacking the constitutive androstane receptor
P50
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P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Sandrine Ménard
@ast
Sandrine Ménard
@en
Sandrine Ménard
@es
Sandrine Ménard
@nl
Sandrine Ménard
@sl
type
label
Sandrine Ménard
@ast
Sandrine Ménard
@en
Sandrine Ménard
@es
Sandrine Ménard
@nl
Sandrine Ménard
@sl
prefLabel
Sandrine Ménard
@ast
Sandrine Ménard
@en
Sandrine Ménard
@es
Sandrine Ménard
@nl
Sandrine Ménard
@sl
P106
P21
P31
P496
0000-0001-8867-199X