about
Ischemic stroke injury is mediated by aberrant Cdk5Pharmacological enhancement of mGlu5 receptors rescues behavioral deficits in SHANK3 knock-out mice.A53T-alpha-synuclein overexpression impairs dopamine signaling and striatal synaptic plasticity in old miceA2A adenosine receptor antagonism enhances synaptic and motor effects of cocaine via CB1 cannabinoid receptor activationc-Jun N-terminal kinase has a key role in Alzheimer disease synaptic dysfunction in vivo.Mechanisms underlying the impairment of hippocampal long-term potentiation and memory in experimental Parkinson's disease.Cellular localization of TRPC3 channel in rat brain: preferential distribution to oligodendrocytes.Trace amines depress GABA B response in dopaminergic neurons by inhibiting G-betagamma-gated inwardly rectifying potassium channels.Endogenous 17β-estradiol is required for activity-dependent long-term potentiation in the striatum: interaction with the dopaminergic systemAlpha-Synuclein Produces Early Behavioral Alterations via Striatal Cholinergic Synaptic Dysfunction by Interacting With GluN2D N-Methyl-D-Aspartate Receptor Subunit.Neo-synthesis of estrogenic or androgenic neurosteroids determine whether long-term potentiation or depression is induced in hippocampus of male rat.Dopamine-containing neurons are silenced by energy deprivation: a defensive response or beginning of cell death?Persistent activation of microglia and NADPH oxidase [corrected] drive hippocampal dysfunction in experimental multiple sclerosis.Plastic abnormalities in experimental Huntington's disease.Dopamine-mediated regulation of corticostriatal synaptic plasticity.Plasticity and repair in the post-ischemic brain.Short-term and long-term plasticity at corticostriatal synapses: implications for learning and memory.Modulatory role of androgenic and estrogenic neurosteroids in determining the direction of synaptic plasticity in the CA1 hippocampal region of male rats.Direct and indirect pathways of basal ganglia: a critical reappraisal.Interaction between basal ganglia and limbic circuits in learning and memory processes.Interferon-β1a protects neurons against mitochondrial toxicity via modulation of STAT1 signaling: electrophysiological evidence.Inhibition of phosphodiesterases rescues striatal long-term depression and reduces levodopa-induced dyskinesia.Erratum: Persistent activation of microglia and NADPH oxidase drive hippocampal dysfunction in experimental multiple sclerosis.Electrophysiology and pharmacology of striatal neuronal dysfunction induced by mitochondrial complex I inhibition.A2A adenosine receptor antagonists protect the striatum against rotenone-induced neurotoxicity.Electrophysiological actions of zonisamide on striatal neurons: Selective neuroprotection against complex I mitochondrial dysfunction.Neural 17β-estradiol facilitates long-term potentiation in the hippocampal CA1 region.Ischemic-LTP in striatal spiny neurons of both direct and indirect pathway requires the activation of D1-like receptors and NO/soluble guanylate cyclase/cGMP transmission.Induction of heat shock protein 70 reduces the alteration of striatal electrical activity caused by mitochondrial impairment.Involvement of transient receptor potential-like channels in responses to mGluR-I activation in midbrain dopamine neurons.Depression of mGluR-mediated IPSCs by 5-HT in dopamine neurons of the rat substantia nigra pars compacta.Transient receptor potential-like channels mediate metabotropic glutamate receptor EPSCs in rat dopamine neurones.Decreased NR2B subunit synaptic levels cause impaired long-term potentiation but not long-term depression.Protective role of hydrogen peroxide in oxygen-deprived dopaminergic neurones of the rat substantia nigra.Epilepsy, amyloid-β, and D1 dopamine receptors: a possible pathogenetic link?Different synaptic stimulation patterns influence the local androgenic and estrogenic neurosteroid availability triggering hippocampal synaptic plasticity in the male rat.Interferon-β1a modulates glutamate neurotransmission in the CNS through CaMKII and GluN2A-containing NMDA receptors.Na+/Ca2+ exchanger maintains ionic homeostasis in the peri-infarct area.Rebalance of striatal NMDA/AMPA receptor ratio underlies the reduced emergence of dyskinesia during D2-like dopamine agonist treatment in experimental Parkinson's disease.Abnormal cortical lysosomal β-hexosaminidase and β-galactosidase activity at post-synaptic sites during Alzheimer's disease progression.
P50
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P50
description
researcher
@en
wetenschapper
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հետազոտող
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name
Alessandro Tozzi
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Alessandro Tozzi
@en
Alessandro Tozzi
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Alessandro Tozzi
@nl
Alessandro Tozzi
@sl
type
label
Alessandro Tozzi
@ast
Alessandro Tozzi
@en
Alessandro Tozzi
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Alessandro Tozzi
@nl
Alessandro Tozzi
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prefLabel
Alessandro Tozzi
@ast
Alessandro Tozzi
@en
Alessandro Tozzi
@es
Alessandro Tozzi
@nl
Alessandro Tozzi
@sl
P106
P1153
7006852996
P21
P2798
P31
P496
0000-0003-3153-2424