about
Diaryl Disulfides as Novel Stabilizers of Tumor Suppressor Pdcd4Attenuation of the ELAV1-like protein HuR sensitizes adenocarcinoma cells to the intrinsic apoptotic pathway by increasing the translation of caspase-2L.Development of a high-throughput cell-based reporter assay to identify stabilizers of tumor suppressor Pdcd4.A New Immunomodulatory Role for Peroxisomes in Macrophages Activated by the TLR4 Ligand Lipopolysaccharide.Tricyclic guanidine alkaloids from the marine sponge Acanthella cavernosa that stabilize the tumor suppressor PDCD4.Inflammation-induced loss of Pdcd4 is mediated by phosphorylation-dependent degradationActinopolysporins A-C and tubercidin as a Pdcd4 stabilizer from the halophilic actinomycete Actinopolyspora erythraea YIM 90600HIF-1alpha and p53 as targets of NO in affecting cell proliferation, death and adaptation.HIF-1 and p53: communication of transcription factors under hypoxia.Tristetraprolin regulation of interleukin-22 productionTumor hypoxia and cancer progression.Downregulation of programmed cell death 4 by inflammatory conditions contributes to the generation of the tumor promoting microenvironmentCryptocaryols A-H, α-pyrone-containing 1,3-polyols from Cryptocarya sp. implicated in stabilizing the tumor suppressor Pdcd4.MicroRNA-27b contributes to lipopolysaccharide-mediated peroxisome proliferator-activated receptor gamma (PPARgamma) mRNA destabilization.Tumour stroma-derived lipocalin-2 promotes breast cancer metastasis.Induction of Glucocorticoid-induced Leucine Zipper (GILZ) Contributes to Anti-inflammatory Effects of the Natural Product Curcumin in Macrophages.Suppression of hypoxia-inducible factor-1α contributes to the antiangiogenic activity of red propolis polyphenols in human endothelial cells.A combination of hypoxia and lipopolysaccharide activates tristetraprolin to destabilize proinflammatory mRNAs such as tumor necrosis factor-alpha.Tumor necrosis factor-alpha causes accumulation of a ubiquitinated form of hypoxia inducible factor-1alpha through a nuclear factor-kappaB-dependent pathway.Hypoxia Causes Downregulation of Dicer in Hepatocellular Carcinoma, Which Is Required for Upregulation of Hypoxia-Inducible Factor 1α and Epithelial-Mesenchymal Transition.NO restores HIF-1alpha hydroxylation during hypoxia: role of reactive oxygen species.p300 relieves p53-evoked transcriptional repression of hypoxia-inducible factor-1 (HIF-1).PI3K/Akt is required for heat shock proteins to protect hypoxia-inducible factor 1alpha from pVHL-independent degradation.Roles of hypoxia-inducible factor-1alpha (HIF-1alpha) versus HIF-2alpha in the survival of hepatocellular tumor spheroids.Erratum: Tristetraprolin regulation of interleukin-22 production.Glucocorticoid-induced leucine zipper is downregulated in human alveolar macrophages upon Toll-like receptor activation.Lymphotoxin α, a novel target of posttranscriptional gene regulation by HuR in HepG2 cells.Macrophages attenuate the transcription of CYP1A1 in breast tumor cells and enhance their proliferationLights on for low oxygen: a noninvasive mouse model useful for sensing oxygen deficiencyHIF-1α protein is upregulated in HIF-2α depleted cells via enhanced translationuORF-Tools-Workflow for the determination of translation-regulatory upstream open reading framesTranslation of TNFAIP2 is tightly controlled by upstream open reading framesMacrophage HIF-2α regulates tumor-suppressive Spint1 in the tumor microenvironment
P50
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P50
description
onderzoeker
@nl
researcher
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ricercatore
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հետազոտող
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name
Tobias Schmid
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Tobias Schmid
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Tobias Schmid
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Tobias Schmid
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Tobias Schmid
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type
label
Tobias Schmid
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Tobias Schmid
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Tobias Schmid
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Tobias Schmid
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Tobias Schmid
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prefLabel
Tobias Schmid
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Tobias Schmid
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Tobias Schmid
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Tobias Schmid
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Tobias Schmid
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P214
P106
P21
P214
P31
P496
0000-0002-1952-5259
P6770
P734
P735
P7859
viaf-70009739