Advances in therapeutic strategies for Leber’s hereditary optic neuropathy

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Advances in therapeutic strategies for Leber’s hereditary optic neuropathy

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wetenschappelijk artikel

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наукова стаття, опублікована в листопаді 2015

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Advances in therapeutic strategies for Leber’s hereditary optic neuropathy

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Advances in therapeutic strategies for Leber’s hereditary optic neuropathy

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Advances in therapeutic strategies for Leber’s hereditary optic neuropathy

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Advances in therapeutic strategies for Leber’s hereditary optic neuropathy

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Advances in therapeutic strategies for Leber’s hereditary optic neuropathy

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Advances in therapeutic strategies for Leber’s hereditary optic neuropathy

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P1476

Advances in therapeutic strategies for Leber’s hereditary optic neuropathy

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P2093

Alfredo A. Sadun

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Rustum Karanjia

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Making heads or tails of phospholipids in mitochondria

Mitochondrial dysfunction as a cause of optic neuropathies

Mitochondrial DNA mutation associated with Leber's hereditary optic neuropathy

The mitochondrial-targeted compound SS-31 re-energizes ischemic mitochondria by interacting with cardiolipin

Gene delivery to mitochondria by targeting modified adenoassociated virus suppresses Leber's hereditary optic neuropathy in a mouse model.

Neuroprotective effects of intravitreal mesenchymal stem cell transplantation in experimental glaucoma.

Reduction of early reperfusion injury with the mitochondria-targeting peptide bendavia

α-Tocotrienol quinone modulates oxidative stress response and the biochemistry of aging.

Leber hereditary optic neuropathy

New therapies for optic neuropathies: development in experimental models.

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1439-1446

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scholarly article

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10.1517/21678707.2015.1098531

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P433

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P478

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2015-11-11T00:00:00Z

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