about
Constitutive phosphorylation of aurora-a on ser51 induces its stabilization and consequent overexpression in cancerOncogenic role of nuclear accumulated Aurora-A.Selective inhibition of unfolded protein response induces apoptosis in pancreatic cancer cells.The hypoxia-inducible epigenetic regulators Jmjd1a and G9a provide a mechanistic link between angiogenesis and tumor growth.Down-regulation of Cdk inhibitor p27 in oral squamous cell carcinoma.Periostin promotes invasion and anchorage-independent growth in the metastatic process of head and neck cancer.Interaction between von Hippel-Lindau Protein and Fatty Acid Synthase Modulates Hypoxia Target Gene Expression.Aurora-B/AIM-1 kinase activity is involved in Ras-mediated cell transformation.Overexpression of Aurora-A potentiates HRAS-mediated oncogenic transformation and is implicated in oral carcinogenesis.Transfection of p27(Kip1) threonine residue 187 mutant type gene, which is not influenced by ubiquitin-mediated degradation, induces cell cycle arrest in oral squamous cell carcinoma cells.Hypoxia-inducible factor-1α promotes cell survival during ammonia stress response in ovarian cancer stem-like cells.Hypoxia-inducible factor-2 alpha up-regulates CD70 under hypoxia and enhances anchorage-independent growth and aggressiveness in cancer cells.
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Shojiro Kitajima
@ast
Shojiro Kitajima
@en
Shojiro Kitajima
@es
Shojiro Kitajima
@nl
Shojiro Kitajima
@sl
type
label
Shojiro Kitajima
@ast
Shojiro Kitajima
@en
Shojiro Kitajima
@es
Shojiro Kitajima
@nl
Shojiro Kitajima
@sl
prefLabel
Shojiro Kitajima
@ast
Shojiro Kitajima
@en
Shojiro Kitajima
@es
Shojiro Kitajima
@nl
Shojiro Kitajima
@sl
P108
P106
P31
P496
0000-0002-4000-0473