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Bacterial RTX toxins allow acute ATP release from human erythrocytes directly through the toxin pore.Python erythrocytes are resistant to α-hemolysin from Escherichia coli.Vasopressin-independent targeting of aquaporin-2 by selective E-prostanoid receptor agonists alleviates nephrogenic diabetes insipidus.Intact colonic KCa1.1 channel activity in KCNMB2 knockout mice.Primary cilium-dependent sensing of urinary flow and paracrine purinergic signaling.Inhibition of P2X Receptors Protects Human Monocytes against Damage by Leukotoxin from Aggregatibacter actinomycetemcomitans and α-Hemolysin from Escherichia coliLoop Diuretics Diminish Hemolysis Induced by α-Hemolysin from Escherichia coli.P2X1, P2X4, and P2X7 Receptor Knock Out Mice Expose Differential Outcome of Sepsis Induced by α-Haemolysin Producing Escherichia coli[Ca2+]i Oscillations and IL-6 Release Induced by α-Hemolysin from Escherichia coli Require P2 Receptor Activation in Renal Epithelia.Sialic acid residues are essential for cell lysis mediated by leukotoxin from Aggregatibacter actinomycetemcomitans.Renal epithelial cells can release ATP by vesicular fusionP2X Receptors Inhibit NaCl Absorption in mTAL Independently of Nitric Oxide.P2Y2 receptor knock-out mice display normal NaCl absorption in medullary thick ascending limb.P2X receptors trigger intracellular alkalization in isolated perfused mouse medullary thick ascending limb.Inhibition of the sarco/endoplasmic reticulum (ER) Ca2+-ATPase by thapsigargin analogs induces cell death via ER Ca2+ depletion and the unfolded protein response.Comment on "Aggregatibacter actinomycetemcomitans-induced hypercitrullination links periodontal infection to autoimmunity in rheumatoid arthritis".Leukotoxin from Aggregatibacter actinomycetemcomitans causes shrinkage and P2X receptor-dependent lysis of human erythrocytes.Erythrocyte P2X receptor expression is correlated with change in haematocrit in patients admitted to the ICU with blood pathogen-positive sepsisFurosemide-induced urinary acidification is caused by pronounced H+ secretion in the thick ascending limbHyperaldosteronism after decreased renal K+ excretion in KCNMB2 knockout miceα-Haemolysin production, as a single factor, causes fulminant sepsis in a model of Escherichia coli-induced bacteraemiaP2X1 receptor blockers reduce the number of circulating thrombocytes and the overall survival of urosepsis with haemolysin-producing Escherichia coli
P50
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P50
description
onderzoeker
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researcher
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հետազոտող
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name
Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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type
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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Helle A Praetorius
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P106
P31
P496
0000-0002-0970-5061