about
Role of epigenetics in the etiology of germ cell cancerIL-37 requires the receptors IL-18Rα and IL-1R8 (SIGIRR) to carry out its multifaceted anti-inflammatory program upon innate signal transductionSegregation of Naturally Occurring Mitochondrial DNA Variants in a Mini-Pig ModelNext-generation sequence analysis of cancer xenograft models.New Monoclonal Antibodies to Defined Cell Surface Proteins on Human Pluripotent Stem Cells.Seminoma and embryonal carcinoma footprints identified by analysis of integrated genome-wide epigenetic and expression profiles of germ cell cancer cell linesGenomic characterisation of small cell lung cancer patient-derived xenografts generated from endobronchial ultrasound-guided transbronchial needle aspiration specimens.BTB-ZF transcriptional regulator PLZF modifies chromatin to restrain inflammatory signaling programs.A Synthetic Lethal Interaction between Glutathione Synthesis and Mitochondrial Reactive Oxygen Species Provides a Tumor-Specific Vulnerability Dependent on STAT3.FGFR2 mutation in 46,XY sex reversal with craniosynostosisGut microbial metabolites limit the frequency of autoimmune T cells and protect against type 1 diabetes.A Versatile Strategy for Isolating a Highly Enriched Population of Intestinal Stem Cells.Sustained Low-Dose Treatment with the Histone Deacetylase Inhibitor LBH589 Induces Terminal Differentiation of Osteosarcoma CellsPUMA promotes apoptosis of hematopoietic progenitors driving leukemic progression in a mouse model of myelodysplasia.Haplotyping the human leukocyte antigen system from single chromosomes.Impact of endurance exercise training on adipocyte microRNA expression in overweight men.Overcoming Monocarboxylate Transporter 8 (MCT8)-Deficiency to Promote Human Oligodendrocyte Differentiation and Myelination.Muscle Stem Cells Undergo Extensive Clonal Drift during Tissue Growth via Meox1-Mediated Induction of G2 Cell-Cycle Arrest.Comprehensive characterization of distinct states of human naive pluripotency generated by reprogramming.Fine Tuning of Canonical Wnt Stimulation Enhances Differentiation Of Pluripotent Stem Cells Independent of β-Catenin-Mediated TCF Signaling.The tumor suppressor Hic1 maintains chromosomal stability independent of Tp53.Transient and Permanent Reconfiguration of Chromatin and Transcription Factor Occupancy Drive Reprogramming.Cell Type of Origin Dictates the Route to Pluripotency.Low-Dose Histone Deacetylase Inhibitor Treatment Leads to Tumor Growth Arrest and Multi-Lineage Differentiation of Malignant Rhabdoid Tumors.Transcriptional profile in afferent lymph cells following vaccination with liposomes incorporating CpG.TrawlerWeb: an online de novo motif discovery tool for next-generation sequencing datasets.Deep multi-region whole-genome sequencing reveals heterogeneity and gene-by-environment interactions in treatment-naive, metastatic lung cancerIdentification of dynamic undifferentiated cell states within the male germlineInhibition of activin signaling in lung adenocarcinoma increases the therapeutic index of platinum chemotherapyGILZ-dependent modulation of mTORC1 regulates spermatogonial maintenance
P50
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P50
description
onderzoeker
@nl
researcher
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հետազոտող
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name
Fernando J Rossello
@ast
Fernando J Rossello
@en
Fernando J Rossello
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Fernando J Rossello
@nl
Fernando J Rossello
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type
label
Fernando J Rossello
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Fernando J Rossello
@en
Fernando J Rossello
@es
Fernando J Rossello
@nl
Fernando J Rossello
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prefLabel
Fernando J Rossello
@ast
Fernando J Rossello
@en
Fernando J Rossello
@es
Fernando J Rossello
@nl
Fernando J Rossello
@sl
P106
P1153
55633615900
P31
P496
0000-0003-3885-8777