Spatial, Temporal, and Functional Assessment of LC3-Dependent Autophagy in Shigella flexneri Dissemination

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Spatial, Temporal, and Functional Assessment of LC3-Dependent Autophagy in Shigella flexneri Dissemination

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im August 2018 veröffentlichter wissenschaftlicher Artikel

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wetenschappelijk artikel

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наукова стаття, опублікована в серпні 2018

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Spatial, Temporal, and Functio ...... higella flexneri Dissemination

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Spatial, Temporal, and Functio ...... higella flexneri Dissemination

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Spatial, Temporal, and Functio ...... higella flexneri Dissemination

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Spatial, Temporal, and Functio ...... higella flexneri Dissemination

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Spatial, Temporal, and Functio ...... higella flexneri Dissemination

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Spatial, Temporal, and Functio ...... higella flexneri Dissemination

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P1476

Spatial, Temporal, and Functio ...... higella flexneri Dissemination

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Erin Weddle

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Hervé Agaisse

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Structurally distinct bacterial TBC-like GAPs link Arf GTPase to Rab1 inactivation to counteract host defenses

p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

p62 and NDP52 proteins target intracytosolic Shigella and Listeria to different autophagy pathways

Atg8 controls phagophore expansion during autophagosome formation.

Activation of the CDC42 effector N-WASP by the Shigella flexneri IcsA protein promotes actin nucleation by Arp2/3 complex and bacterial actin-based motility

Molecular and Cellular Mechanisms of Shigella flexneri Dissemination.

The Shigella flexneri type 3 secretion system is required for tyrosine kinase-dependent protrusion resolution, and vacuole escape during bacterial dissemination

Regulation mechanisms and signaling pathways of autophagy

The TBK1 adaptor and autophagy receptor NDP52 restricts the proliferation of ubiquitin-coated bacteria

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scholarly article

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10.1128/IAI.00134-18

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English

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2018-08-01T00:00:00Z

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29844234

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immunology

Shigella flexneri

parasitology

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6056868

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