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Annexin A1 and the Resolution of Inflammation: Modulation of Neutrophil Recruitment, Apoptosis, and ClearanceTumor Necrosis Factor, but Not Neutrophils, Alters the Metabolic Profile in Acute Experimental ArthritisPhosphatidylinositol 3-Kinase γ is required for the development of experimental cerebral malaria.Switching off key signaling survival molecules to switch on the resolution of inflammation.Annexin A1 Is Involved in the Resolution of Inflammatory Responses during Leishmania braziliensis Infection.Induction of eosinophil apoptosis by hydrogen peroxide promotes the resolution of allergic inflammationImpairment of stress granule assembly via inhibition of the eIF2alpha phosphorylation sensitizes glioma cells to chemotherapeutic agents.Inhibition of Phosphodiesterase-4 during Pneumococcal Pneumonia Reduces Inflammation and Lung Injury in Mice.Plasmin and plasminogen induce macrophage reprogramming and regulate key steps of inflammation resolution via annexin A1.Dengue-3 encephalitis promotes anxiety-like behavior in mice.The role and effects of glucocorticoid-induced leucine zipper in the context of inflammation resolution.Annexin A1 modulates natural and glucocorticoid-induced resolution of inflammation by enhancing neutrophil apoptosis.The resolution of acute inflammation induced by cyclic AMP is dependent on annexin A1.Proresolving Actions of Synthetic and Natural Protease Inhibitors Are Mediated by Annexin A1.PI3Kγ deficiency enhances seizures severity and associated outcomes in a mouse model of convulsions induced by intrahippocampal injection of pilocarpine.Altered intracellular signaling cascades in peripheral blood mononuclear cells from BD patients.Annexin A1 promotes timely resolution of inflammation in murine gout.Plasmin induces in vivo monocyte recruitment through protease-activated receptor-1-, MEK/ERK-, and CCR2-mediated signaling.Angiotensin-(1-7) and Alamandine Promote Anti-inflammatory Response in Macrophages andMediators of the Resolution of the Inflammatory ResponsePlasminogen and the Plasminogen Receptor, Plg-RKT, Regulate Macrophage Phenotypic, and Functional ChangesCyclic AMP Regulates Key Features of Macrophages via PKA: Recruitment, Reprogramming and EfferocytosisInhibition of the sphingosine-1-phosphate pathway promotes the resolution of neutrophilic inflammation
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description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Juliana P Vago
@ast
Juliana P Vago
@en
Juliana P Vago
@es
Juliana P Vago
@nl
Juliana P Vago
@sl
type
label
Juliana P Vago
@ast
Juliana P Vago
@en
Juliana P Vago
@es
Juliana P Vago
@nl
Juliana P Vago
@sl
prefLabel
Juliana P Vago
@ast
Juliana P Vago
@en
Juliana P Vago
@es
Juliana P Vago
@nl
Juliana P Vago
@sl
P106
P21
P31
P496
0000-0001-8188-3738