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Endogenous Parkin Preserves Dopaminergic Substantia Nigral Neurons following Mitochondrial DNA Mutagenic StressMitochondrial DNA damage in a mouse model of Alzheimer's disease decreases amyloid beta plaque formationIncrease in muscle mitochondrial biogenesis does not prevent muscle loss but increased tumor size in a mouse model of acute cancer-induced cachexiaStriatal dysfunctions associated with mitochondrial DNA damage in dopaminergic neurons in a mouse model of Parkinson's diseaseMitochondrial disease: mtDNA and protein segregation mysteries in iPSCsThe roles of PINK1, parkin, and mitochondrial fidelity in Parkinson's diseaseCOX2-derived primary and cyclopentenone prostaglandins are increased after asphyxial cardiac arrest.Transient systemic mtDNA damage leads to muscle wasting by reducing the satellite cell poolDisruption of Bax protein prevents neuronal cell death but produces cognitive impairment in mice following traumatic brain injury.The role of cytochrome c oxidase deficiency in ROS and amyloid plaque formation.What role does mitochondrial stress play in neurodegenerative diseases?Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent manner.Methods to detect mitophagy in neurons during diseaseEGR1 recruits TET1 to shape the brain methylome during development and upon neuronal activity
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description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Alicia M Pickrell
@ast
Alicia M Pickrell
@en
Alicia M Pickrell
@es
Alicia M Pickrell
@nl
Alicia M Pickrell
@sl
type
label
Alicia M Pickrell
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Alicia M Pickrell
@en
Alicia M Pickrell
@es
Alicia M Pickrell
@nl
Alicia M Pickrell
@sl
prefLabel
Alicia M Pickrell
@ast
Alicia M Pickrell
@en
Alicia M Pickrell
@es
Alicia M Pickrell
@nl
Alicia M Pickrell
@sl
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0000-0002-8470-6056