about
IL-1α reversibly inhibits skeletal muscle ryanodine receptor. a novel mechanism for critical illness myopathy?Evidence that the EphA2 receptor exacerbates ischemic brain injury.Human skeletal muscle plasmalemma alters its structure to change its Ca2+-handling following heavy-load resistance exercise.Store-operated Ca²⁺ entry is not required for store refilling in skeletal muscle.Development of a high-throughput method for real-time assessment of cellular metabolism in intact long skeletal muscle fibre bundles.Leaky ryanodine receptors delay the activation of store overload-induced Ca2+ release, a mechanism underlying malignant hyperthermia-like events in dystrophic muscle.Doublet stimulation increases Ca2+ binding to troponin C to ensure rapid force development in skeletal muscle.NADPH oxidase mediates microtubule alterations and diaphragm dysfunction in dystrophic mice.NHE- and diffusion-dependent proton fluxes across the tubular system membranes of fast-twitch muscle fibers of the rat.A quantitative description of tubular system Ca(2+) handling in fast- and slow-twitch muscle fibresChanges in plasma membrane Ca-ATPase and stromal interacting molecule 1 expression levels for Ca2+ signaling in dystrophic mdx mouse muscleUltra-rapid activation and deactivation of store-operated Ca2+ entry in skeletal muscleUpregulation of store-operated Ca2+ entry in dystrophic mdx mouse muscleLongitudinal and transversal propagation of excitation along the tubular system of rat fast-twitch muscle fibres studied by high speed confocal microscopyActivation and propagation of Ca2+ release from inside the sarcoplasmic reticulum network of mammalian skeletal muscle
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onderzoeker
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հետազոտող
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Tanya R Cully
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P106
P31
P496
0000-0002-7047-9913