about
Carbon black nanoparticles induce type II epithelial cells to release chemotaxins for alveolar macrophages.Antiviral potential of cathelicidinsInflammatory Bowel Disease Drugs: A Focus on AutophagyAntiviral activity and increased host defense against influenza infection elicited by the human cathelicidin LL-37Cationic host defense peptides; novel antimicrobial therapeutics against Category A pathogens and emerging infections.A candidate H1N1 pandemic influenza vaccine elicits protective immunity in mice.Waddlia chondrophila infects and multiplies in ovine trophoblast cells stimulating an inflammatory immune response.Improved protection against avian influenza H5N1 virus by a single vaccination with virus-like particles in skin using microneedlesMAPK Activation Is Essential for Waddlia chondrophila Induced CXCL8 Expression in Human Epithelial Cells.Secondary necrosis of apoptotic neutrophils induced by the human cathelicidin LL-37 is not proinflammatory to phagocytosing macrophagesOvine trophoblasts express cathelicidin host defence peptide in response to infection.Nanoparticle interactions with zinc and iron: implications for toxicology and inflammation.Carbon Nanoparticles Inhibit the Antimicrobial Activities of the Human Cathelicidin LL-37 through Structural Alteration.Cathelicidins display conserved direct antiviral activity towards rhinovirus.Serum exposed to nanoparticle carbon black displays increased potential to induce macrophage migration.The human cathelicidin LL-37 preferentially promotes apoptosis of infected airway epithelium.The human cationic host defense peptide LL-37 mediates contrasting effects on apoptotic pathways in different primary cells of the innate immune system.NO-loaded Zn(2+)-exchanged zeolite materials: a potential bifunctional anti-bacterial strategy.The type III intermediate filament vimentin regulates organelle distribution and modulates autophagyAntiviral Host Defence PeptidesSecondary necrosis of apoptotic neutrophils induced by the human cathelicidin LL-37 is not proinflammatory to phagocytosing macrophagesReduced alveolar macrophage migration induced by acute ambient particle (PM10) exposureProinflammatory Effects of Particles on Macrophages and Epithelial CellsThe Inflammatory Bowel Disease Drug Azathioprine Induces Autophagy via mTORC1 and the Unfolded Protein Response Sensor PERKInteractions Between Autophagy and the Unfolded Protein Response: Implications for Inflammatory Bowel DiseaseAntiviral therapeutic approaches for human rhinovirus infectionsCitrullination Alters the Antiviral and Immunomodulatory Activities of the Human Cathelicidin LL-37 During Rhinovirus Infection
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Peter G Barlow
@ast
Peter G Barlow
@en
Peter G Barlow
@es
Peter G Barlow
@nl
type
label
Peter G Barlow
@ast
Peter G Barlow
@en
Peter G Barlow
@es
Peter G Barlow
@nl
prefLabel
Peter G Barlow
@ast
Peter G Barlow
@en
Peter G Barlow
@es
Peter G Barlow
@nl
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P31
P496
0000-0002-6516-9312