about
Inhibition of catechol-O-methyltransferase (COMT) in the brain does not affect the action of dopamine and levodopa: an in vitro electrophysiological evidence from rat mesencephalic dopamine neuronsDysfunctional dopaminergic neurotransmission in asocial BTBR mice.Dopamine neuronal loss contributes to memory and reward dysfunction in a model of Alzheimer's disease.Trace amines depress D(2)-autoreceptor-mediated responses on midbrain dopaminergic cells.Blockade of nociceptin/orphanin FQ receptor signaling in rat substantia nigra pars reticulata stimulates nigrostriatal dopaminergic transmission and motor behavior.Trace amines depress GABA B response in dopaminergic neurons by inhibiting G-betagamma-gated inwardly rectifying potassium channels.Dopamine-containing neurons are silenced by energy deprivation: a defensive response or beginning of cell death?Functional alterations of the dopaminergic and glutamatergic systems in spontaneous α-synuclein overexpressing rats.On the effects of psychostimulants, antidepressants, and the antiparkinsonian drug levodopa on dopamine neurons.Paradoxical abatement of striatal dopaminergic transmission by cocaine and methylphenidatePINK1 heterozygous mutations induce subtle alterations in dopamine-dependent synaptic plasticity.Loss of striatal cannabinoid CB1 receptor function in attention-deficit / hyperactivity disorder mice with point-mutation of the dopamine transporter.Role of aberrant striatal dopamine D1 receptor/cAMP/protein kinase A/DARPP32 signaling in the paradoxical calming effect of amphetamine.Dopamine selectively reduces GABA(B) transmission onto dopaminergic neurones by an unconventional presynaptic action.Actions of methylphenidate on dopaminergic neurons of the ventral midbrain.Oxidative stress in stroke pathophysiology validation of hydrogen peroxide metabolism as a pharmacological target to afford neuroprotection.Differential effect of carbamazepine and oxcarbazepine on excitatory synaptic transmission in rat hippocampus.Dopaminergic dysfunction is associated with IL-1β-dependent mood alterations in experimental autoimmune encephalomyelitis.Electrophysiologic changes in ventral midbrain dopaminergic neurons resulting from (+/-) -3,4-methylenedioxymethamphetamine (MDMA-"Ecstasy").Modification of levodopa responses by deprenyl (selegiline): an electrophysiological and behavioral study in the rat relevant to Parkinson's disease.Whole cell patch-clamp recordings of rat midbrain dopaminergic neurons isolate a sulphonylurea- and ATP-sensitive component of potassium currents activated by hypoxia.Tranylcypromine, but not moclobemide, prolongs the inhibitory action of dopamine on midbrain dopaminergic neurons: an in vitro electrophysiological study3,4-Methylenedioxymethamphetamine (MDMA) Alters Synaptic Dopamine Release in the Dorsal Striatum Through Nicotinic Receptors and DAT InhibitionAuthor Correction: Blunting neuroinflammation with resolvin D1 prevents early pathology in a rat model of Parkinson's diseaseThe striatal-enriched protein Rhes is a critical modulator of cocaine-induced molecular and behavioral responsesBlunting neuroinflammation with resolvin D1 prevents early pathology in a rat model of Parkinson's disease
P50
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description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Mauro Federici
@ast
Mauro Federici
@en
Mauro Federici
@es
Mauro Federici
@nl
type
label
Mauro Federici
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Mauro Federici
@en
Mauro Federici
@es
Mauro Federici
@nl
altLabel
mauro federici
@en
prefLabel
Mauro Federici
@ast
Mauro Federici
@en
Mauro Federici
@es
Mauro Federici
@nl
P106
P1153
7006237367
P31
P496
0000-0003-4563-1677