Double-walled carbon nanotubes trigger IL-1β release in human monocytes through Nlrp3 inflammasome activation
about
Advances in mechanisms and signaling pathways of carbon nanotube toxicitySintered indium-tin oxide particles induce pro-inflammatory responses in vitro, in part through inflammasome activationInvestigation of the pulmonary bioactivity of double-walled carbon nanotubesFibrosis biomarkers in workers exposed to MWCNTsThe complex cascade of cellular events governing inflammasome activation and IL-1β processing in response to inhaled particlesLung inflammation caused by inhaled toxicants: a reviewBiological interactions of carbon-based nanomaterials: From coronation to degradation.An Allergic Lung Microenvironment Suppresses Carbon Nanotube-Induced Inflammasome Activation via STAT6-Dependent Inhibition of Caspase-1Stealth filaments: Polymer chain length and conformation affect the in vivo fate of PEGylated potato virus X.Mechanisms of lung fibrosis induced by carbon nanotubes: towards an Adverse Outcome Pathway (AOP)The NLRP3 inflammasome in pathogenic particle and fibre-associated lung inflammation and diseasesPre-clinical immunotoxicity studies of nanotechnology-formulated drugs: Challenges, considerations and strategyCytokines as biomarkers of nanoparticle immunotoxicityCarbon Nanotubes and Chronic Granulomatous DiseaseThe mechanism of pleural inflammation by long carbon nanotubes: interaction of long fibres with macrophages stimulates them to amplify pro-inflammatory responses in mesothelial cellsThe alarmin IL-1α is a master cytokine in acute lung inflammation induced by silica micro- and nanoparticlesExposure to nickel oxide nanoparticles induces pulmonary inflammation through NLRP3 inflammasome activation in ratsImpact of carbon nanotubes and graphene on immune cellsInflammasome activation in airway epithelial cells after multi-walled carbon nanotube exposure mediates a profibrotic response in lung fibroblastsAutophagy and lysosomal dysfunction as emerging mechanisms of nanomaterial toxicitySilver nanoparticles induce degradation of the endoplasmic reticulum stress sensor activating transcription factor-6 leading to activation of the NLRP-3 inflammasomeEnvironmental impact of multi-wall carbon nanotubes in a novel model of exposure: systemic distribution, macrophage accumulation, and amyloid depositionActivation of Proinflammatory Responses in Cells of the Airway Mucosa by Particulate Matter: Oxidant- and Non-Oxidant-Mediated Triggering MechanismsInnate Immune Responses to Nanoparticle Exposure in the LungSuppression of ribosomal function triggers innate immune signaling through activation of the NLRP3 inflammasomePhagolysosome Acidification is Required for Silica and Engineered Nanoparticle-induced Lysosome Membrane Permeabilization and Resultant NLRP3 Inflammasome ActivityIncreasing both CoCrMo-alloy particle size and surface irregularity induces increased macrophage inflammasome activation in vitro potentially through lysosomal destabilization mechanismsImmunological responses triggered by photothermal therapy with carbon nanotubes in combination with anti-CTLA-4 therapy to inhibit cancer metastasis.Chitosan functionalisation of gold nanoparticles encourages particle uptake and induces cytotoxicity and pro-inflammatory conditions in phagocytic cells, as well as enhancing particle interactions with serum componentsSynergistic Costimulatory Effect of Chlamydia pneumoniae with Carbon Nanoparticles on NLRP3 Inflammasome-Mediated Interleukin-1β Secretion in Macrophages.Nanomedicine and nanotoxicology: the pros and cons for neurodegeneration and brain cancer.The role of Rho-kinases in IL-1β release through phagocytosis of fibrous particles in human monocytes.Characterization of Potential Exposures to Nanoparticles and Fibers during Manufacturing and Recycling of Carbon Nanotube Reinforced Polypropylene Composites.A Single Aspiration of Rod-like Carbon Nanotubes Induces Asbestos-like Pulmonary Inflammation Mediated in Part by the IL-1 Receptor.Mechanisms of carbon nanotube-induced pulmonary fibrosis: a physicochemical characteristic perspective.Pulmonary surfactant protein SP-D opsonises carbon nanotubes and augments their phagocytosis and subsequent pro-inflammatory immune response.Reactive oxygen species trigger NF-κB-mediated NLRP3 inflammasome activation induced by zinc oxide nanoparticles in A549 cells.Iron-related toxicity of single-walled carbon nanotubes and crocidolite fibres in human mesothelial cells investigated by Synchrotron XRF microscopy.Polymeric Nanoparticles Induce NLRP3 Inflammasome Activation and Promote Breast Cancer Metastasis.Assessing NLRP3 Inflammasome Activation by Nanoparticles.
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P2860
Double-walled carbon nanotubes trigger IL-1β release in human monocytes through Nlrp3 inflammasome activation
description
im November 2011 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published on 16 November 2011
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в серпні 2012
@uk
name
Double-walled carbon nanotubes ...... Nlrp3 inflammasome activation
@en
Double-walled carbon nanotubes ...... Nlrp3 inflammasome activation
@nl
type
label
Double-walled carbon nanotubes ...... Nlrp3 inflammasome activation
@en
Double-walled carbon nanotubes ...... Nlrp3 inflammasome activation
@nl
prefLabel
Double-walled carbon nanotubes ...... Nlrp3 inflammasome activation
@en
Double-walled carbon nanotubes ...... Nlrp3 inflammasome activation
@nl
P2093
P50
P1476
Double-walled carbon nanotubes ...... Nlrp3 inflammasome activation
@en
P2093
Christophe Dardenne
Etienne Meunier
José Bernad
Maryse Béraud
P304
P356
10.1016/J.NANO.2011.11.004
P577
2011-11-16T00:00:00Z