about
Cell type-specific plasticity of striatal projection neurons in parkinsonism and L-DOPA-induced dyskinesia.Chemogenetic stimulation of striatal projection neurons modulates responses to Parkinson's disease therapy.Mechanisms of dopamine D1 receptor-mediated ERK1/2 activation in the parkinsonian striatum and their modulation by metabotropic glutamate receptor type 5.Pathophysiology of L-dopa-induced motor and non-motor complications in Parkinson's disease.Gene expression analyses identify Narp contribution in the development of L-DOPA-induced dyskinesia.Extracellular signal-regulated kinase activation is required for consolidation and reconsolidation of memory at an early stage of ontogenesis.L-DOPA activates ERK signaling and phosphorylates histone H3 in the striatonigral medium spiny neurons of hemiparkinsonian mice.Mitogen- and stress-activated protein kinase 1 is required for specific signaling responses in dopamine-denervated mouse striatum, but is not necessary for L-DOPA-induced dyskinesia.Gα(olf) mutation allows parsing the role of cAMP-dependent and extracellular signal-regulated kinase-dependent signaling in L-3,4-dihydroxyphenylalanine-induced dyskinesia.
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description
hulumtuese
@sq
researcher
@en
wetenschapper
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հետազոտող
@hy
name
Cristina Alcacer
@ast
Cristina Alcacer
@en
Cristina Alcacer
@es
Cristina Alcacer
@nl
type
label
Cristina Alcacer
@ast
Cristina Alcacer
@en
Cristina Alcacer
@es
Cristina Alcacer
@nl
prefLabel
Cristina Alcacer
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Cristina Alcacer
@en
Cristina Alcacer
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Cristina Alcacer
@nl
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0000-0003-2502-0983