about
Estrogen-dependent regulation of human uterine natural killer cells promotes vascular remodelling via secretion of CCL2.A novel mouse model of endometriosis mimics human phenotype and reveals insights into the inflammatory contribution of shed endometriumRelevant human tissue resources and laboratory models for use in endometriosis research.Elevated peritoneal expression and estrogen regulation of nociceptive ion channels in endometriosis.Estrogen receptor (ER) agonists differentially regulate neuroangiogenesis in peritoneal endometriosis via the repellent factor SLIT3.Estradiol is a critical mediator of macrophage-nerve cross talk in peritoneal endometriosis.ERβ-dependent effects on uterine endothelial cells are cell specific and mediated via Sp1.Cortisol regulates the paracrine action of macrophages by inducing vasoactive gene expression in endometrial cells.EP2 receptor antagonism reduces peripheral and central hyperalgesia in a preclinical mouse model of endometriosis.Cyclin E is recruited to the nuclear matrix during differentiation, but is not recruited in cancer cells.Cancer-associated variant expression and interaction of CIZ1 with cyclin A1 in differentiating male germ cells.The Regulation of Ciz1 Gene Expression During Mouse DevelopmentEffects of macrophage depletion on characteristics of cervix remodeling and pregnancy in CD11b-dtr mice
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description
hulumtuese
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հետազոտող
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Erin Greaves
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Erin Greaves
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Erin Greaves
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P21
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0000 0004 2681 6796
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P496
0000-0001-9165-5851