Eckols reduce dental pulp inflammation through the ERK1/2 pathway independent of COX-2 inhibition

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Schisandrin C enhances mitochondrial biogenesis and autophagy in C2C12 skeletal muscle cells: potential involvement of anti-oxidative mechanisms.Orally Administered Phlorotannins from Suppress Chemical Mediator Release and Cyclooxygenase-2 Signaling to Alleviate Mouse Ear Swelling

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Eckols reduce dental pulp inflammation through the ERK1/2 pathway independent of COX-2 inhibition

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наукова стаття, опублікована в серпні 2014

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Eckols reduce dental pulp infl ...... ndependent of COX-2 inhibition

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Eckols reduce dental pulp infl ...... ndependent of COX-2 inhibition

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Eckols reduce dental pulp infl ...... ndependent of COX-2 inhibition

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Eckols reduce dental pulp infl ...... ndependent of COX-2 inhibition

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Eckols reduce dental pulp infl ...... ndependent of COX-2 inhibition

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Eckols reduce dental pulp infl ...... ndependent of COX-2 inhibition

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Eckols reduce dental pulp infl ...... ndependent of COX-2 inhibition

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B-S Yun

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H-K Yi

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N-H Park

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P-H Hwang

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T-H Kwon

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U Paudel

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Y-H Lee

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A new phloroglucinol derivative from the brown alga Eisenia bicyclis: potential for the effective treatment of diabetic complications.

Invasion of dentinal tubules by oral bacteria.

Dentin matrix protein 1 and phosphate homeostasis are critical for postnatal pulp, dentin and enamel formation.

Inflammatory and immunological aspects of dental pulp repair.

Vital pulp therapy-current progress of dental pulp regeneration and revascularization

Neuroprotective effect of edible brown alga Eisenia bicyclis on amyloid beta peptide-induced toxicity in PC12 cells.

Anti-inflammatory activity of edible brown alga Eisenia bicyclis and its constituents fucosterol and phlorotannins in LPS-stimulated RAW264.7 macrophages.

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scholarly article

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10.1111/ODI.12266

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2014-08-01T00:00:00Z

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24924779

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inflammation

Eckols reduce dental pulp inflammation through the ERK1/2 pathway independent of COX-2 inhibition

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Eckols reduce dental pulp inflammation through the ERK1/2 pathway independent of COX-2 inhibition

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