about
Uracil DNA glycosylase BKRF3 contributes to Epstein-Barr virus DNA replication through physical interactions with proteins in viral DNA replication complex.The ESCRT machinery is recruited by the viral BFRF1 protein to the nucleus-associated membrane for the maturation of Epstein-Barr Virus.Constitutive and ligand-induced EGFR signalling triggers distinct and mutually exclusive downstream signalling networks.BGLF4 kinase modulates the structure and transport preference of the nuclear pore complex to facilitate nuclear import of Epstein-Barr virus lytic proteinsEpstein-barr virus, the immune system, and associated diseasesMaintenance of Epstein-Barr Virus Latent Status by a Novel Mechanism, Latent Membrane Protein 1-Induced Interleukin-32, via the Protein Kinase Cδ Pathway.Conserved herpesvirus kinases target the DNA damage response pathway and TIP60 histone acetyltransferase to promote virus replication.Role of the TSG101 gene in Epstein-Barr virus late gene transcription.Epstein-Barr virus protein kinase BGLF4 targets the nucleus through interaction with nucleoporins.BCL10GFP fusion protein as a substrate for analysis of determinants required for mucosa-associated lymphoid tissue 1 (MALT1)-mediated cleavage.Epstein-Barr virus BGLF4 kinase suppresses the interferon regulatory factor 3 signaling pathwayAutocrine CCL3 and CCL4 induced by the oncoprotein LMP1 promote Epstein-Barr virus-triggered B cell proliferation.Protein array identification of substrates of the Epstein-Barr virus protein kinase BGLF4OmpA is the critical component for Escherichia coli invasion-induced astrocyte activation.Effect of phosphorylation on the transactivation activity of Epstein-Barr virus BMRF1, a major target of the viral BGLF4 kinase.Delineation of a 16 amino acid sequence that forms a core DNA recognition motif in the Epstein-Barr virus EBNA-1 protein.Novel expression and regulation of TIMP-1 in Epstein Barr virus-infected cells and its impact on cell survival.Epstein-Barr virus LMP2A suppresses MHC class II expression by regulating the B-cell transcription factors E47 and PU.1.Hepatitis C virus NS4A inhibits cap-dependent and the viral IRES-mediated translation through interacting with eukaryotic elongation factor 1A.Detection of Epstein-Barr virus BGLF4 protein kinase in virus replication compartments and virus particles.
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Mei-Ru Chen
@ast
Mei-Ru Chen
@en
Mei-Ru Chen
@es
Mei-Ru Chen
@nl
type
label
Mei-Ru Chen
@ast
Mei-Ru Chen
@en
Mei-Ru Chen
@es
Mei-Ru Chen
@nl
prefLabel
Mei-Ru Chen
@ast
Mei-Ru Chen
@en
Mei-Ru Chen
@es
Mei-Ru Chen
@nl
P106
P1153
7406356172
P31
P496
0000-0002-7007-8036