about
Cerebral malaria: mysteries at the blood-brain barrierAdaptive immunity against Leishmania nucleoside hydrolase maps its c-terminal domain as the target of the CD4+ T cell-driven protective responseSilencing cytokeratin 18 gene inhibits intracellular replication of Trypanosoma cruzi in HeLa cells but not binding and invasion of trypanosomesHost Resistance to Plasmodium-Induced Acute Immune Pathology Is Regulated by Interleukin-10 Receptor Signaling.CD8+ T cells and IFN-γ mediate the time-dependent accumulation of infected red blood cells in deep organs during experimental cerebral malaria.Interferons and interferon regulatory factors in malariaA rapid and robust tri-color flow cytometry assay for monitoring malaria parasite development.A pathogenic role for CD4+ T cells during Chikungunya virus infection in mice.Inflammatory Flt3l is essential to mobilize dendritic cells and for T cell responses during Plasmodium infection.Plasmodium vivax: restricted tropism and rapid remodeling of CD71-positive reticulocytesThe CTLA-4 and PD-1/PD-L1 inhibitory pathways independently regulate host resistance to Plasmodium-induced acute immune pathology.Immunization with the MAEBL M2 Domain Protects against Lethal Plasmodium yoelii Infection.Ex Vivo Maturation Assay for Testing Antimalarial Sensitivity of Rodent Malaria Parasites.Brain microvessel cross-presentation is a hallmark of experimental cerebral malaria.Rodent Plasmodium-infected red blood cells: imaging their fates and interactions within their hosts.Pathogenic CD8+ T cells in experimental cerebral malaria.Adaptive immunity is essential in preventing recrudescence of Plasmodium yoelii malaria parasites after artesunate treatment.Measuring antigen presentation in mouse brain endothelial cells ex vivo and in vitro.Tissue-Resident CD169(+) Macrophages Form a Crucial Front Line against Plasmodium Infection.Spatiotemporal requirements for IRF7 in mediating type I IFN-dependent susceptibility to blood-stage Plasmodium infection.Strain-specific protective immunity following vaccination against experimental Trypanosoma cruzi infection.Cutting edge: Clec9A+ dendritic cells mediate the development of experimental cerebral malaria.Co-infection with Chikungunya virus alters trafficking of pathogenic CD8(+) T cells into the brain and prevents Plasmodium-induced neuropathology.Dual modal ultra-bright nanodots with aggregation-induced emission and gadolinium-chelation for vascular integrity and leakage detection.CD41 is a reliable identification and activation marker for murine basophils in the steady state and during helminth and malarial infections.Interferon regulatory factor 1 is essential for pathogenic CD8+ T cell migration and retention in the brain during experimental cerebral malaria.Protective Immunity Against Trypanosoma cruzi Infection in a Highly Susceptible Mouse Strain After Vaccination with Genes Encoding the Amastigote Surface Protein-2 and Trans-SialidasePlasmodium co-infection protects against chikungunya virus-induced pathologiesImmunologically relevant strain polymorphism in the Amastigote Surface Protein 2 of Trypanosoma cruziLung endothelial cell antigen cross-presentation to CD8+T cells drives malaria-associated lung injuryAuthor Correction: Lung endothelial cell antigen cross-presentation to CD8+T cells drives malaria-associated lung injury
P50
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P50
description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Carla Claser
@ast
Carla Claser
@en
Carla Claser
@es
Carla Claser
@nl
type
label
Carla Claser
@ast
Carla Claser
@en
Carla Claser
@es
Carla Claser
@nl
prefLabel
Carla Claser
@ast
Carla Claser
@en
Carla Claser
@es
Carla Claser
@nl
P106
P21
P31
P496
0000-0002-5395-9632