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Common data elements for spinal cord injury clinical research: a National Institute for Neurological Disorders and Stroke projectRecommendations for the National Institute for Neurologic Disorders and Stroke spinal cord injury common data elements for children and youth with SCI.Developing a data sharing community for spinal cord injury research.Aberrant sensory responses are dependent on lesion severity after spinal cord contusion injury in mice.TGF-alpha increases astrocyte invasion and promotes axonal growth into the lesion following spinal cord injury in mice.Strategies for spinal cord injury repair.Sensory stimulation prior to spinal cord injury induces post-injury dysesthesia in mice.Independent evaluation of the anatomical and behavioral effects of Taxol in rat models of spinal cord injury.Injured mice at the gym: review, results and considerations for combining chondroitinase and locomotor exercise to enhance recovery after spinal cord injuryProgranulin expression is upregulated after spinal contusion in mice.Transforming growth factor α transforms astrocytes to a growth-supportive phenotype after spinal cord injuryCharacterization and distribution of putative 5-ht7 receptors in guinea-pig brainA reassessment of a classic neuroprotective combination therapy for spinal cord injured rats: LPS/pregnenolone/indomethacinModulating Sema3A signal with a L1 mimetic peptide is not sufficient to promote motor recovery and axon regeneration after spinal cord injury.Alterations in chondroitin sulfate proteoglycan expression occur both at and far from the site of spinal contusion injury.Robust axonal growth and a blunted macrophage response are associated with impaired functional recovery after spinal cord injury in the MRL/MpJ mouseElevated MMP-9 in the lumbar cord early after thoracic spinal cord injury impedes motor relearning in mice.In the presence of danger: The extracellular matrix defensive response to central nervous system injury.Harmonization of Databases: A Step for Advancing the Knowledge About Spinal Cord Injury.Large animal and primate models of spinal cord injury for the testing of novel therapies.Differentiation of substantia gelatinosa-like regions in intraspinal and intracerebral transplants of embryonic spinal cord tissue in the rat.CRMP3 is required for hippocampal CA1 dendritic organization and plasticity.The interaction of RS 25259-197, a potent and selective antagonist, with 5-HT3 receptors, in vitroL1 cell adhesion molecule is essential for the maintenance of hyperalgesia after spinal cord injury.A novel tetrodotoxin-sensitive, voltage-gated sodium channel expressed in rat and human dorsal root ganglia.Distribution of radioiodinated recombinant human nerve growth factor in primate brain following intracerebroventricular infusion.Corticospinal tract transection reduces H-reflex circadian rhythm in rats.Translational Stroke Research: Vision and Opportunities.Enhanced axonal growth into a spinal cord contusion injury site in a strain of mouse (129X1/SvJ) with a diminished inflammatory response.Epidural Spinal Stimulation to Improve Bladder, Bowel, and Sexual Function in Individuals With Spinal Cord Injuries: A Framework for Clinical Research.Bone marrow transplants provide tissue protection and directional guidance for axons after contusive spinal cord injury in rats.Structure and function of a novel voltage-gated, tetrodotoxin-resistant sodium channel specific to sensory neurons.Brain-derived neurotrophic factor stimulates hindlimb stepping and sprouting of cholinergic fibers after spinal cord injury.Mice lacking L1 cell adhesion molecule have deficits in locomotion and exhibit enhanced corticospinal tract sprouting following mild contusion injury to the spinal cord.Pegylated brain-derived neurotrophic factor shows improved distribution into the spinal cord and stimulates locomotor activity and morphological changes after injury.The interaction of a new motor skill and an old one: H-reflex conditioning and locomotion in rats.Operant conditioning of H-reflex can correct a locomotor abnormality after spinal cord injury in rats.Recommendations for evaluation of neurogenic bladder and bowel dysfunction after spinal cord injury and/or disease
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P50
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researcher
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wetenschapper
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հետազոտող
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Lyn B Jakeman
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Lyn B Jakeman
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Lyn B Jakeman
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Lyn B Jakeman
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Lyn B Jakeman
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Lyn B Jakeman
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Lyn B Jakeman
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Lyn B Jakeman
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Lyn Jakeman
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Lyn B Jakeman
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Lyn B Jakeman
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Lyn B Jakeman
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Lyn B Jakeman
@nl
P106
P1153
7003497928
P31
P496
0000-0002-1760-8819