SOCS2 deletion protects against hepatic steatosis but worsens insulin resistance in high-fat-diet-fed mice
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Implication of inflammatory signaling pathways in obesity-induced insulin resistanceLipid profiling and transcriptomic analysis reveals a functional interplay between estradiol and growth hormone in liver.Deficiency of the transcriptional repressor B cell lymphoma 6 (Bcl6) is accompanied by dysregulated lipid metabolismDevelopment and Validation of a Three-gene Prognostic Signature for Patients with Hepatocellular CarcinomaFOXO1 content is reduced in cystic fibrosis and increases with IGF-I treatment.Genome-wide analysis of hepatic lipid content in extreme obesityCytochrome P450 1B1: An unexpected modulator of liver fatty acid homeostasisDepletion of suppressor of cytokine signaling-1a causes hepatic steatosis and insulin resistance in zebrafish.Fish oil alleviated high-fat diet-induced non-alcoholic fatty liver disease via regulating hepatic lipids metabolism and metaflammation: a transcriptomic study.Estrogens regulate the hepatic effects of growth hormone, a hormonal interplay with multiple fates.Type I interferons and microbial metabolites of tryptophan modulate astrocyte activity and central nervous system inflammation via the aryl hydrocarbon receptor.The influence of estrogens on the biological and therapeutic actions of growth hormone in the liver.The suppressor of cytokine signalling 2 (SOCS2), traumatic brain injury and microglial/macrophage regulationSuppressor of cytokine signalling (SOCS) proteins as guardians of inflammatory responses critical for regulating insulin sensitivity.SOCS Proteins in Macrophage Polarization and Function.The ubiquitin ligase Cullin5SOCS2 regulates NDR1/STK38 stability and NF-κB transactivation.Involvement of Cbl-b-mediated macrophage inactivation in insulin resistance.SOCS2 inhibited mitochondria biogenesis via inhibiting p38 MAPK/ATF2 pathway in C2C12 cells.Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy.Nutritional strategy to prevent fatty liver and insulin resistance independent of obesity by reducing glucose-dependent insulinotropic polypeptide responses in mice.Intrauterine hyperglycemia exposure results in intergenerational inheritance via DNA methylation reprogramming on F1 PGCs.
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SOCS2 deletion protects against hepatic steatosis but worsens insulin resistance in high-fat-diet-fed mice
description
im August 2012 veröffentlichter wissenschaftlicher Artikel
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scientific article published on 04 May 2012
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wetenschappelijk artikel
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наукова стаття, опублікована в серпні 2012
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name
SOCS2 deletion protects agains ...... ance in high-fat-diet-fed mice
@en
SOCS2 deletion protects agains ...... ance in high-fat-diet-fed mice
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type
label
SOCS2 deletion protects agains ...... ance in high-fat-diet-fed mice
@en
SOCS2 deletion protects agains ...... ance in high-fat-diet-fed mice
@nl
prefLabel
SOCS2 deletion protects agains ...... ance in high-fat-diet-fed mice
@en
SOCS2 deletion protects agains ...... ance in high-fat-diet-fed mice
@nl
P2093
P50
P356
P1433
P1476
SOCS2 deletion protects agains ...... ance in high-fat-diet-fed mice
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P2093
Fahad Zadjali
Irene Hernandez-Hernandez
Mercedes Mirecki-Garrido
Paolo Parini
Ruyman Santana-Farre
P304
P356
10.1096/FJ.12-205583
P407
P50
P577
2012-05-04T00:00:00Z