about
KCa2 channels activation prevents [Ca2+]i deregulation and reduces neuronal death following glutamate toxicity and cerebral ischemiaTargeting pathogen metabolism without collateral damage to the hostIdentification and characterization of a novel, shorter isoform of the small conductance Ca2+ -activated K+ channel SK2Inhibition of the AIF/CypA complex protects against intrinsic death pathways induced by oxidative stress.Subcellular expression and neuroprotective effects of SK channels in human dopaminergic neuronsFibril polymorphism affects immobilized non-amyloid flanking domains of huntingtin exon1 rather than its polyglutamine coreRNA editing in the central cavity as a mechanism to regulate surface expression of the voltage-gated potassium channel Kv1.1.Small-conductance Ca2+-activated potassium type 2 channels regulate the formation of contextual fear memory.The metalloprotease-disintegrin ADAM8 contributes to temozolomide chemoresistance and enhanced invasiveness of human glioblastoma cells.Protective Roles for Potassium SK/K(Ca)2 Channels in Microglia and Neurons.Design of a novel thiophene inhibitor of 15-lipoxygenase-1 with both anti-inflammatory and neuroprotective propertiesInflammation and NF-kappaB in Alzheimer's disease and diabetes.Statins: mechanisms of neuroprotection.BID links ferroptosis to mitochondrial cell death pathways.Statins--increasing or reducing the risk of Parkinson's disease?K(Ca)2 and k(ca)3 channels in learning and memory processes, and neurodegeneration.Regulators of mitochondrial Ca(2+) homeostasis in cerebral ischemia.Activation of SK2 channels preserves ER Ca²⁺ homeostasis and protects against ER stress-induced cell death.SK channel activation modulates mitochondrial respiration and attenuates neuronal HT-22 cell damage induced by H2O2.The serine protease inhibitor TLCK attenuates intrinsic death pathways in neurons upstream of mitochondrial demise.Novel N-phenyl-substituted thiazolidinediones protect neural cells against glutamate- and tBid-induced toxicity.α1-antitrypsin modulates microglial-mediated neuroinflammation and protects microglial cells from amyloid-β-induced toxicity.SK2 channels regulate mitochondrial respiration and mitochondrial Ca2+ uptake.Small conductance Ca2+-activated K+ channels in the plasma membrane, mitochondria and the ER: Pharmacology and implications in neuronal diseases.The role of Ca2+ in cell death caused by oxidative glutamate toxicity and ferroptosis.Impedance measurement for real time detection of neuronal cell death.KBP interacts with SCG10, linking Goldberg-Shprintzen syndrome to microtubule dynamics and neuronal differentiation.Neuronal AKAP150 coordinates PKA and Epac-mediated PKB/Akt phosphorylation.Inhibition of HIF-prolyl-4-hydroxylases prevents mitochondrial impairment and cell death in a model of neuronal oxytosis.Lithium protects hippocampal progenitors, cognitive performance and hypothalamus-pituitary function after irradiation to the juvenile rat brain.Pretreatment with lovastatin prevents N-methyl-D-aspartate-induced neurodegeneration in the magnocellular nucleus basalis and behavioral dysfunction.Activation of KCNN3/SK3/K(Ca)2.3 channels attenuates enhanced calcium influx and inflammatory cytokine production in activated microglia.Trifluoperazine rescues human dopaminergic cells from wild-type α-synuclein-induced toxicity.Bcl-xL knockout attenuates mitochondrial respiration and causes oxidative stress that is compensated by pentose phosphate pathway activity.TNF-alpha-mediates neuroprotection against glutamate-induced excitotoxicity via NF-kappaB-dependent up-regulation of K2.2 channels.Lovastatin induces neuroprotection through tumor necrosis factor receptor 2 signaling pathways.Interleukin-6 upregulates neuronal adenosine A1 receptors: implications for neuromodulation and neuroprotection.Glucose-regulated protein 75 determines ER-mitochondrial coupling and sensitivity to oxidative stress in neuronal cells.Mitochondrial Ca2+-activated K+ channels and their role in cell life and death pathways.One protein, different cell fate: the differential outcome of depleting GRP75 during oxidative stress in neurons.
P50
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P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Amalia Dolga
@ast
Amalia Dolga
@en
Amalia Dolga
@es
Amalia Dolga
@nl
type
label
Amalia Dolga
@ast
Amalia Dolga
@en
Amalia Dolga
@es
Amalia Dolga
@nl
altLabel
A. Dolga
@nl
prefLabel
Amalia Dolga
@ast
Amalia Dolga
@en
Amalia Dolga
@es
Amalia Dolga
@nl
P1006
P214
P1006
P106
P21
P213
0000 0003 9089 9189
P214
P31
P496
0000-0001-5400-5614
P569
1978-01-01T00:00:00Z
P735
P7859
viaf-284584006