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Cancer Microenvironment: What Can We Learn from the Stem Cell NicheIntercellular crosstalk in human malignant melanoma.Ageing as an Important Risk Factor for Cancer.Pharmacological activation of estrogen receptors-α and -β differentially modulates keratinocyte differentiation with functional impact on wound healingEmerging role of tissue lectins as microenvironmental effectors in tumors and wounds.Ecology of melanoma cell.Revelation of fibroblast protein commonalities and differences and their possible roles in wound healing and tumourigenesis using co-culture models of cells.Cultivation-dependent plasticity of melanoma phenotype.Upregulation of IL-6, IL-8 and CXCL-1 production in dermal fibroblasts by normal/malignant epithelial cells in vitro: Immunohistochemical and transcriptomic analyses.Fibroblasts prepared from different types of malignant tumors stimulate expression of luminal marker keratin 8 in the EM-G3 breast cancer cell line.Phosphorylated human lectin galectin-3: analysis of ligand binding by histochemical monitoring of normal/malignant squamous epithelia and by isothermal titration calorimetry.Mouse 3T3 fibroblasts under the influence of fibroblasts isolated from stroma of human basal cell carcinoma acquire properties of multipotent stem cells.Synthetic polyamine BPA-C8 inhibits TGF-β1-mediated conversion of human dermal fibroblast to myofibroblasts and establishment of galectin-1-rich extracellular matrix in vitro.How Signaling Molecules Regulate Tumor Microenvironment: Parallels to Wound Repair.Phenotypic characterization of human keratinocytes in coculture reveals differential effects of fibroblasts from benign fibrous histiocytoma (dermatofibroma) as compared to cells from its malignant form and to normal fibroblasts.Phenotypic characterization of oral mucosa: what is normal?Microenvironment‑driven resistance to B‑Raf inhibition in a melanoma patient is accompanied by broad changes of gene methylation and expression in distal fibroblasts.ER-α agonist induces conversion of fibroblasts into myofibroblasts, while ER-β agonist increases ECM production and wound tensile strength of healing skin wounds in ovariectomised rats.Smooth muscle actin-expressing stromal fibroblasts in head and neck squamous cell carcinoma: Increased expression of galectin-1 and induction of poor prognosis factorsHuman Galectins Induce Conversion of Dermal Fibroblasts into Myofibroblasts and Production of Extracellular Matrix: Potential Application in Tissue Engineering and Wound RepairCancer-associated fibroblasts are not formed from cancer cells by epithelial-to-mesenchymal transition in nu/nu mice
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description
biolog
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onderzoeker
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researcher
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հետազոտող
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name
Pavol Szabo
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Pavol Szabo
@cs
Pavol Szabo
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Pavol Szabo
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Pavol Szabo
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type
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Pavol Szabo
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Pavol Szabo
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Pavol Szabo
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Pavol Szabo
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Pavol Szabo
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Pavol Szabo
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Pavol Szabo
@cs
Pavol Szabo
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Pavol Szabo
@es
Pavol Szabo
@nl
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0000-0002-1944-1493
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