about
In vivo confocal neuroimaging (ICON): non-invasive, functional imaging of the mammalian CNS with cellular resolution.Electrical brain stimulation induces dendritic stripping but improves survival of silent neurons after optic nerve damage.Mechanistic understanding of nanoparticles' interactions with extracellular matrix: the cell and immune systemPlease keep calm: investigating hippocampal function without stress.Vision restoration after brain and retina damage: the "residual vision activation theory".Brain restoration as an emerging field in neurology and neuroscience.Cytotoxicity and apoptotic gene expression in an in vitro model of the blood-brain barrier following exposure to poly(butylcyanoacrylate) nanoparticles.Cholinergic Potentiation of Restoration of Visual Function after Optic Nerve Damage in Rats.Toxicity of polymeric nanoparticles in vivo and in vitro.Brain-state-dependent non-invasive brain stimulation and functional priming: a hypothesisExperience-dependent plasticity and vision restoration in rats after optic nerve crush.Four different types of protease-activated receptors are widely expressed in the brain and are up-regulated in hippocampus by severe ischemia.Increase of prothrombin-mRNA after global cerebral ischemia in rats, with constant expression of protease nexin-1 and protease-activated receptors.Transient focal ischemia in rat brain differentially regulates mRNA expression of protease-activated receptors 1 to 4.In vivo visualisation of nanoparticle entry into central nervous system tissue.Transcorneal alternating current stimulation after severe axon damage in rats results in "long-term silent survivor" neurons.Increase in proliferation and gliogenesis but decrease of early neurogenesis in the rat forebrain shortly after transient global ischemia.Non-invasive electrical brain stimulation: from acute to late-stage treatment of central nervous system damage.Surfactants, not size or zeta-potential influence blood-brain barrier passage of polymeric nanoparticles.(S)-4C3HPG reduces infarct size after focal cerebral ischemia.Oral application of carbon nanofibers in rats increases blood concentration of IL6 and IL10 and decreases locomotor activity.Repetitive Transcorneal Alternating Current Stimulation Reduces Brain Idling State After Long-term Vision Loss.Transcorneal alternating current stimulation induces EEG "aftereffects" only in rats with an intact visual system but not after severe optic nerve damage.Predictive value of changes in electroencephalogram and excitatory postsynaptic field potential for CA1 damage after global ischaemia in rats.Preclinical model of transcorneal alternating current stimulation in freely moving rats.No improvement of functional and histological outcome after application of the metabotropic glutamate receptor 5 agonist CHPG in a model of endothelin-1-induced focal ischemia in rats.Functional protection of learning and memory abilities in rats with vascular dementia.Recovery of axonal transport after partial optic nerve damage is associated with secondary retinal ganglion cell death in vivo.Distinct influence of the group III metabotropic glutamate receptor agonist (R,S)-4-phosphonophenylglycine [(R,S)-PPG] on different forms of neuronal damageMajor effects on blood-retina barrier passage by minor alterations in design of polybutylcyanoacrylate nanoparticlesEvaluation of Toxicity and Neural Uptake In Vitro and In Vivo of Superparamagnetic Iron Oxide NanoparticlesClenbuterol protects mouse cerebral cortex and rat hippocampus from ischemic damage and attenuates glutamate neurotoxicity in cultured hippocampal neurons by induction of NGFFocal ischemia induces expression of protease-activated receptor1 (PAR1) and PAR3 on microglia and enhances PAR4 labeling in the penumbra
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description
hulumtuese
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researcher
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հետազոտող
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name
Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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Petra Henrich-Noack
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