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Risk of Bacterial Vaginosis Among Women With Herpes Simplex Virus Type 2 Infection: A Systematic Review and Meta-analysisIntravaginal Chlamydia trachomatis Challenge Infection Elicits TH1 and TH17 Immune Responses in Mice That Promote Pathogen Clearance and Genital Tract DamageA caveat for T cell transfer studies: generation of cytotoxic anti-Thy1.2 antibodies in Thy1.1 congenic mice given Thy1.2+ tumors or T cellsHuman female genital tract infection by the obligate intracellular bacterium Chlamydia trachomatis elicits robust Type 2 immunity.Limitations of the criteria used to diagnose histologic endometritis in epidemiologic pelvic inflammatory disease researchMedroxyprogesterone acetate impairs human dendritic cell activation and function.Brefeldin A, but not monensin, enables flow cytometric detection of interleukin-4 within peripheral T cells responding to ex vivo stimulation with Chlamydia trachomatis.Dendritic cell activation and memory cell development are impaired among mice administered medroxyprogesterone acetate prior to mucosal herpes simplex virus type 1 infectionTransient detection of Chlamydial-specific Th1 memory cells in the peripheral circulation of women with history of Chlamydia trachomatis genital tract infection.Use of transcriptional profiling to delineate the initial response of mice to intravaginal herpes simplex virus type 2 infection.Fluorescent labeling reliably identifies Chlamydia trachomatis in living human endometrial cells and rapidly and accurately quantifies chlamydial inclusion forming units.Medroxyprogesterone acetate and levonorgestrel increase genital mucosal permeability and enhance susceptibility to genital herpes simplex virus type 2 infection.Dendritic cell function and pathogen-specific T cell immunity are inhibited in mice administered levonorgestrel prior to intranasal Chlamydia trachomatis infectionIL-4-secreting eosinophils promote endometrial stromal cell proliferation and prevent Chlamydia-induced upper genital tract damage.HIV and Hormonal Contraception: Bench and Bedside.Setting Sights on Chlamydia Immunity's Central Paradigm: Can We Hit a Moving Target?Exogenous oestrogen inhibits genital transmission of cell-associated HIV-1 in DMPA-treated humanized mice.Delayed processing of blood increases the frequency of activated CD11b+ CD15+ granulocytes which inhibit T cell functionLevonorgestrel and Female Genital Tract Immunity: Time for a Closer LookNorethisterone Enanthate Increases Mouse Susceptibility to Genital Infection with Herpes Simplex Virus Type 2 and HIV Type 1Depot-medroxyprogesterone acetate reduces genital cell-cell adhesion molecule expression and increases genital herpes simplex virus type 2 infection susceptibility in a dose-dependent fashion
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description
researcher
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wetenschapper
@nl
հետազոտող
@hy
name
Rodolfo D Vicetti Miguel
@ast
Rodolfo D Vicetti Miguel
@en
Rodolfo D Vicetti Miguel
@es
Rodolfo D Vicetti Miguel
@nl
type
label
Rodolfo D Vicetti Miguel
@ast
Rodolfo D Vicetti Miguel
@en
Rodolfo D Vicetti Miguel
@es
Rodolfo D Vicetti Miguel
@nl
prefLabel
Rodolfo D Vicetti Miguel
@ast
Rodolfo D Vicetti Miguel
@en
Rodolfo D Vicetti Miguel
@es
Rodolfo D Vicetti Miguel
@nl
P106
P21
P31
P496
0000-0002-2494-8184