about
Globosides but not isoglobosides can impact the development of invariant NKT cells and their interaction with dendritic cellsInvariant NKT cells reduce the immunosuppressive activity of influenza A virus-induced myeloid-derived suppressor cells in mice and humansThe molecular phenotype of endocapillary proliferation: novel therapeutic targets for IgA nephropathy[Glycosphingolipids Gb3 and iGb3. In vivo roles in hemolytic-uremic syndrome and iNKT cell function].Direct acute tubular damage contributes to Shigatoxin-mediated kidney failure.[Globosides as key players in the pathophysiology of Shiga toxin-associated acute kidney failure and Fabry disease].Depletion of globosides and isoglobosides fully reverts the morphologic phenotype of Fabry diseaseHuman renal cell carcinoma induces a dendritic cell subset that uses T-cell crosstalk for tumor-permissive milieu alterations.Renal sulfatides: sphingoid base-dependent localization and region-specific compensation of CerS2-dysfunction.Hepatitis C virus induced endothelial inflammatory response depends on the functional expression of TNFα receptor subtype 2Normal development and function of invariant natural killer T cells in mice with isoglobotrihexosylceramide (iGb3) deficiency.On-Tissue Phospholipase C Digestion for Enhanced MALDI-MS Imaging of Neutral Glycosphingolipids.Sulfatides are required for renal adaptation to chronic metabolic acidosis.ELMO1 protects renal structure and ultrafiltration in kidney development and under diabetic conditions.Thymoma related myasthenia gravis in humans and potential animal models.Myf5 and Myogenin in the development of thymic myoid cells - Implications for a murine in vivo model of myasthenia gravis.A new, robust, and nonradioactive approach for exploring N-myristoylation.Tubular Dickkopf-3 promotes the development of renal atrophy and fibrosis.Modulation of Wnt and Hedgehog signaling pathways is linked to retinoic acid-induced amelioration of chronic allograft dysfunction.Glucosylceramide Synthase Is Involved in Development of Invariant Natural Killer T Cells.Changes in CD73, CD39 and CD26 expression on T-lymphocytes of ANCA-associated vasculitis patients suggest impairment in adenosine generation and turn-over.Recipient Toll-like receptors contribute to chronic graft dysfunction by both MyD88- and TRIF-dependent signaling.cFLIP regulates skin homeostasis and protects against TNF-induced keratinocyte apoptosis.Gene expression fingerprints in human tubulointerstitial inflammation and fibrosis as prognostic markers of disease progression.Adiponectin gene expression and plasma values in obese women during very-low-calorie diet. Relationship with cardiovascular risk factors and insulin resistance.Deficiency of N-myristoylation reveals calcineurin activity as regulator of IFN-γ-producing γδ T cells.Immunosuppression and Aberrant T Cell Development in the Absence of N-Myristoylation.Metabolic Syndrome Negatively Impacts the Outcome of Localized Renal Cell Carcinoma.Male meiotic cytokinesis requires ceramide synthase 3-dependent sphingolipids with unique membrane anchors.Wnt pathway regulation in chronic renal allograft damage.Rhoh deficiency reduces peripheral T-cell function and attenuates allogenic transplant rejectionmRNA-Expression of and Defines Distinct Prognostic Subgroups of Muscle-Invasive Urothelial Bladder Cancer Correlating with Histological VariantsRenal outcomes of STOP-IgAN trial patients in relation to baseline histology (MEST-C scores)Abrogation of nephrotic proteinuria by rituximab treatment in a renal transplant patient with relapsed focal segmental glomerulosclerosisHistiocytosis X and renal insufficiencyNephrotic-range proteinuria following pamidronate therapy in a patient with metastatic breast cancer: mitochondrial toxicity as a pathogenetic concept?Attenuation-based characterization of coronary atherosclerotic plaque: comparison of dual source and dual energy CT with single-source CT and histopathologyThe renal microenvironment modifies dendritic cell phenotypeRenal lipidosis in patients enrolled in a methadone substitution programRapid proteomic analysis for solid tumors reveals LSD1 as a drug target in an end-stage cancer patient
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
S Porubsky
@ast
S Porubsky
@en
S Porubsky
@es
S Porubsky
@nl
type
label
S Porubsky
@ast
S Porubsky
@en
S Porubsky
@es
S Porubsky
@nl
altLabel
Stefan Porubsky
@en
prefLabel
S Porubsky
@ast
S Porubsky
@en
S Porubsky
@es
S Porubsky
@nl
P1053
J-5738-2015
P106
P21
P31
P496
0000-0002-7647-7265