about
Cardiomyocyte-specific expression of CRNK, the C-terminal domain of PYK2, maintains ventricular function and slows ventricular remodeling in a mouse model of dilated cardiomyopathy.Haploinsufficiency of MYBPC3 exacerbates the development of hypertrophic cardiomyopathy in heterozygous mice.Rapid large-scale purification of myofilament proteins using a cleavable His6-tag.Cardiac Myosin-binding Protein C and Troponin-I Phosphorylation Independently Modulate Myofilament Length-dependent Activation.Titin strain contributes to the Frank-Starling law of the heart by structural rearrangements of both thin- and thick-filament proteins.Contractile dysfunction in a mouse model expressing a heterozygous MYBPC3 mutation associated with hypertrophic cardiomyopathy.The naked mole-rat exhibits an unusual cardiac myofilament protein profile providing new insights into heart function of this naturally subterranean rodent.Phosphorylation of cardiac myosin-binding protein-C contributes to calcium homeostasis
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description
onderzoeker
@nl
researcher
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հետազոտող
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name
Mohit Kumar
@ast
Mohit Kumar
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Mohit Kumar
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Mohit Kumar
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মোহিত কুমার
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type
label
Mohit Kumar
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Mohit Kumar
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Mohit Kumar
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Mohit Kumar
@nl
মোহিত কুমার
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prefLabel
Mohit Kumar
@ast
Mohit Kumar
@en
Mohit Kumar
@es
Mohit Kumar
@nl
মোহিত কুমার
@bn
P106
P1153
55547134811
P31
P496
0000-0002-3554-0563