about
The RNA helicase DDX5/p68 is a key factor promoting c-fos expression at different levels from transcription to mRNA exportSplicing switch of an epigenetic regulator by RNA helicases promotes tumor-cell invasivenessThe Ddx5 and Ddx17 RNA helicases are cornerstones in the complex regulatory array of steroid hormone-signaling pathwaysRNA helicases DDX5 and DDX17 dynamically orchestrate transcription, miRNA, and splicing programs in cell differentiationA new advance in alternative splicing databases: from catalogue to detailed analysis of regulation of expression and function of human alternative splicing variants.Splicing factor and exon profiling across human tissues.Splicing programs and cancerAutoimmune mediated regulation of ovarian tumor growth.Cancer-associated SF3B1 mutations affect alternative splicing by promoting alternative branchpoint usage.Alteration of cyclin D1 transcript elongation by a mutated transcription factor up-regulates the oncogenic D1b splice isoform in cancerCoregulators: transducing signal from transcription to alternative splicing.Alternative splicing and breast cancer.DNA damage: RNA-binding proteins protect from near and far.Estrogen regulation and physiopathologic significance of alternative promoters in breast cancer.Mechanistic differences in the activation of estrogen receptor-alpha (ER alpha)- and ER beta-dependent gene expression by cAMP signaling pathway(s).DNA-Damage Response RNA-Binding Proteins (DDRBPs): Perspectives from a New Class of Proteins and Their RNA Targets.Exon-based clustering of murine breast tumor transcriptomes reveals alternative exons whose expression is associated with metastasis.Cotranscriptional splicing potentiates the mRNA production from a subset of estradiol-stimulated genes.Regulation of H-ras splice variant expression by cross talk between the p53 and nonsense-mediated mRNA decay pathways.Follicle-stimulating hormone increases testicular Anti-Mullerian hormone (AMH) production through sertoli cell proliferation and a nonclassical cyclic adenosine 5'-monophosphate-mediated activation of the AMH Gene.Translational reprogramming of colorectal cancer cells induced by 5-fluorouracil through a miRNA-dependent mechanism.Ligand-independent interactions of p160/steroid receptor coactivators and CREB-binding protein (CBP) with estrogen receptor-alpha: regulation by phosphorylation sites in the A/B region depends on other receptor domains.A recently evolved class of alternative 3'-terminal exons involved in cell cycle regulation by topoisomerase inhibitors.
P50
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description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Martin Dutertre
@ast
Martin Dutertre
@en
Martin Dutertre
@es
Martin Dutertre
@nl
type
label
Martin Dutertre
@ast
Martin Dutertre
@en
Martin Dutertre
@es
Martin Dutertre
@nl
prefLabel
Martin Dutertre
@ast
Martin Dutertre
@en
Martin Dutertre
@es
Martin Dutertre
@nl
P106
P1153
6603100191
P31
P496
0000-0003-0278-4120