Progranulin Deficient Mice Develop Nephrogenic Diabetes Insipidus

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Progranulin Deficient Mice Develop Nephrogenic Diabetes Insipidus

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scientific article published on 01 October 2018

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wetenschappelijk artikel

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наукова стаття, опублікована в жовтні 2018

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Progranulin Deficient Mice Develop Nephrogenic Diabetes Insipidus

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Progranulin Deficient Mice Develop Nephrogenic Diabetes Insipidus

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Progranulin Deficient Mice Develop Nephrogenic Diabetes Insipidus

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Progranulin Deficient Mice Develop Nephrogenic Diabetes Insipidus

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Progranulin Deficient Mice Develop Nephrogenic Diabetes Insipidus

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Progranulin Deficient Mice Develop Nephrogenic Diabetes Insipidus

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P1476

Progranulin Deficient Mice Develop Nephrogenic Diabetes Insipidus

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P2093

Annett Wilken-Schmitz

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Irmgard Tegeder

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Jinyang Zeng-Brouwers

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Lucie Valek

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Stefanie Hardt

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The growth factor granulin interacts with cyclin T1 and modulates P-TEFb-dependent transcription

Progranulin: at the interface of neurodegenerative and metabolic diseases

Progranulin gene expression regulates epithelial cell growth and promotes tumor growth in vivo

An extracellular congenital nephrogenic diabetes insipidus mutation of the vasopressin receptor reduces cell surface expression, affinity for ligand, and coupling to the Gs/adenylyl cyclase system

Null mutations in progranulin cause ubiquitin-positive frontotemporal dementia linked to chromosome 17q21

Mutations in progranulin cause tau-negative frontotemporal dementia linked to chromosome 17

Yeast-2-Hybrid data file showing progranulin interactions in human fetal brain and bone marrow libraries.

Exaggerated inflammation, impaired host defense, and neuropathology in progranulin-deficient mice

GSK3beta mediates renal response to vasopressin by modulating adenylate cyclase activity.

Biglycan-triggered TLR-2- and TLR-4-signaling exacerbates the pathophysiology of ischemic acute kidney injury.

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817-830

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scholarly article

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10.14336/AD.2017.1127

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English

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Liliana Schaefer

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2018-10-01T00:00:00Z

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30271659

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6147595

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