about
GABAA receptor-acting neurosteroids: a role in the development and regulation of the stress responseCorticotropin-releasing factor and urocortin differentially modulate rat Purkinje cell dendritic outgrowth and differentiation in vitroThe localisation of urocortin in the adult rat cerebellum: a light and electron microscopic studyRemoval of GABA(A) receptor γ2 subunits from parvalbumin neurons causes wide-ranging behavioral alterations.Neonatal rearing conditions distinctly shape locus coeruleus neuronal activity, dendritic arborization, and sensitivity to corticotrophin-releasing factor.Aberrant location of inhibitory synaptic marker proteins in the hippocampus of dystrophin-deficient mice: implications for cognitive impairment in duchenne muscular dystrophy.Quantitative localisation of synaptic and extrasynaptic GABAA receptor subunits on hippocampal pyramidal cells by freeze-fracture replica immunolabelling.From synapse to behavior: rapid modulation of defined neuronal types with engineered GABAA receptors.During postnatal development endogenous neurosteroids influence GABA-ergic neurotransmission of mouse cortical neurons.Dysfunctional astrocytic and synaptic regulation of hypothalamic glutamatergic transmission in a mouse model of early-life adversity: relevance to neurosteroids and programming of the stress response.Tonic inhibition of accumbal spiny neurons by extrasynaptic α4βδ GABAA receptors modulates the actions of psychostimulants.Extrasynaptic glycine receptors of rodent dorsal raphe serotonergic neurons: a sensitive target for ethanolIndividual domains of Tensin2 exhibit distinct subcellular localisations and migratory effects.Localization of NG2 immunoreactive neuroglia cells in the rat locus coeruleus and their plasticity in response to stress.Immunolocalization of AMPA receptor subunits within the enteric nervous system of the mouse colon and the effect of their activation on spontaneous colonic contractions.Molecular and functional diversity of GABA-A receptors in the enteric nervous system of the mouse colon.Absence of glial α-dystrobrevin causes abnormalities of the blood-brain barrier and progressive brain edema.Differential localization of GABA(A) receptor subunits in relation to rat striatopallidal and pallidopallidal synapses.The postnatal developmental expression pattern of urocortin in the rat olivocerebellar system.Endogenous neurosteroids influence synaptic GABAA receptors during post-natal development.Corticotropin-releasing factor and urocortin regulate spine and synapse formation: structural basis for stress-induced neuronal remodeling and pathology.Corticotropin-releasing factor promotes growth of brain norepinephrine neuronal processes through Rho GTPase regulators of the actin cytoskeleton in rat.Localisation and stress-induced plasticity of GABAA receptor subunits within the cellular networks of the mouse dorsal raphe nucleus.Molecular Characterization of GABA-A Receptor Subunit Diversity within Major Peripheral Organs and Their Plasticity in Response to Early Life Psychosocial Stress.TREK-1 Channel Expression in Smooth Muscle as a Target for Regulating Murine Intestinal Contractility: Therapeutic Implications for Motility Disorders.A GABAergic cell type in the lateral habenula links hypothalamic homeostatic and midbrain motivation circuits with sex steroid signaling.Spatiotemporal Distribution of GABAA Receptor Subunits Within Layer II of Mouse Medial Entorhinal Cortex: Implications for Grid Cell Excitability.Early-life adversity selectively impairs α2-GABA receptor expression in the mouse nucleus accumbens and influences the behavioral effects of cocaineDynamic Modulation of Mouse Locus Coeruleus Neurons by Vasopressin 1a and 1b ReceptorsA Synaptically Connected Hypothalamic Magnocellular Vasopressin-Locus Coeruleus Neuronal Circuit and Its Plasticity in Response to Emotional and Physiological StressGABAA Receptor Subtypes Regulate Stress-Induced Colon Inflammation in MiceEarly-life stress influences acute and sensitized responses of adult mice to cocaine by interacting with GABAA α2 receptor expressionDevelopmental and age-dependent plasticity of GABAA receptors in the mouse colon: Implications in colonic motility and inflammation
P50
Q28241963-67155412-F649-437F-8354-DB8AF48EA29DQ28571592-2CC402E2-F056-4E43-B4E4-DB152B7A1442Q28573768-83C6DFA2-1425-4B08-B805-EF202770CD2CQ30474166-B15F7D24-FC5D-48E2-824E-C60E521557A4Q33802083-1A1D9F5F-7837-4BFC-8334-7107F558B9D7Q35280143-AA8664B9-3FC4-4FBA-8E31-F429905E1D96Q35806141-5BD479E6-8E08-4D29-8316-B1AEEC2639ADQ36166392-4E842A1F-CAB7-453F-A0D4-EF9CE2A73ED4Q36610262-445B18C2-C58B-4145-A9AA-FCE76FC31B27Q37381812-BF23AF09-916E-4A8E-A903-CE62F4176D69Q37479497-17C9CE85-BEEF-4F52-BA80-9271B9A4AFB7Q37642454-DC44E3F8-48B9-4611-9831-6F35EECF2736Q39800672-69C09A14-BFF2-4517-B0E9-BB170A2AF6D0Q39852008-D2DCF3B2-C00D-4374-995E-79055D6093C6Q40004149-F3C5209B-BF8E-4B18-BB49-14194D6F0125Q41857666-5BB66C90-3877-4620-9F48-6AD9CDB0A69CQ42372835-A6C28FA4-4E7E-423D-B429-4EC7F9C811B4Q42482334-9215D873-3020-4F82-8A58-5046FF988899Q47942243-4CB41757-6187-4AB5-9DEA-24FBA11BC716Q48113905-60B075DE-6B9B-45B6-A321-BC9B96FCA87BQ48166124-1FA83C92-BF18-4FF3-9DDD-32B6E9970730Q48372229-EA162457-DB83-40A7-8EB7-FD40DCEF50CAQ48690295-8BBCF78C-0DA7-47FB-A0EC-CFF66D806C8AQ50234147-4B96C581-1D3D-4B19-81C2-F6CB6767CD2CQ52689982-4AC8F15F-D376-4EC6-8B65-43C6F5B6D492Q52730456-3F6B7DEF-3441-4E36-984A-A8A06E3A2D1BQ55449041-999A5C85-ADC9-4FB6-96B0-260473315B85Q58792105-9C195F11-BE7C-4D88-BE4B-639C70C4F1E6Q60961166-1D3D8F95-0DFF-4EB1-AD6F-4A77AE7C02BFQ64070113-D2E43315-BA2F-4BD9-8067-9E5A20E8DEEAQ88869552-1216D8DA-E6DE-4E54-8237-EEBFE33C459DQ91368551-636D6ACE-3006-4896-AE24-1AB1EEDA92BBQ92841225-A92C059B-8D42-4330-BE7B-7CFFA7BBAAE5
P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Jerome D Swinny
@ast
Jerome D Swinny
@en
Jerome D Swinny
@es
Jerome D Swinny
@nl
type
label
Jerome D Swinny
@ast
Jerome D Swinny
@en
Jerome D Swinny
@es
Jerome D Swinny
@nl
prefLabel
Jerome D Swinny
@ast
Jerome D Swinny
@en
Jerome D Swinny
@es
Jerome D Swinny
@nl
P106
P1153
6506457509
P31
P496
0000-0002-8194-5481