about
High-resolution genetic mapping in the diversity outbred mouse population identifies Apobec1 as a candidate gene for atherosclerosis.Metabolic reprogramming through fatty acid transport protein 1 (FATP1) regulates macrophage inflammatory potential and adipose inflammation.Prevention of tumorigenesis in mice by exercise is dependent on strain background and timing relative to carcinogen exposure.Lack of myeloid Fatp1 increases atherosclerotic lesion size in Ldlr-/- mice.Efferocytosis induces a novel SLC program to promote glucose uptake and lactate releaseMyeloid -Deficient Murine Model Revealed Macrophage Activation and Metabolic Phenotype Are Fueled by GLUT1Long-chain acyl-CoA synthetase 1 interacts with key proteins that activate and direct fatty acids into niche hepatic pathwaysDefective fatty acid oxidation in mice with muscle-specific acyl-CoA synthetase 1 deficiency increases amino acid use and impairs muscle function
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Liyang Zhao
@ast
Liyang Zhao
@en
Liyang Zhao
@es
Liyang Zhao
@nl
type
label
Liyang Zhao
@ast
Liyang Zhao
@en
Liyang Zhao
@es
Liyang Zhao
@nl
prefLabel
Liyang Zhao
@ast
Liyang Zhao
@en
Liyang Zhao
@es
Liyang Zhao
@nl
P106
P31
P496
0000-0003-1960-0199