about
Human NKG2D-ligands: cell biology strategies to ensure immune recognitionNatural killer cell cytotoxicity is suppressed by exposure to the human NKG2D ligand MICA*008 that is shed by tumor cells in exosomesMultiple receptors for HLA-G on human natural killer cellsExpression of the UL16 glycoprotein of Human Cytomegalovirus protects the virus-infected cell from attack by natural killer cellsGlycosyl-Phosphatidyl-Inositol (GPI)-Anchors and Metalloproteases: Their Roles in the Regulation of Exosome Composition and NKG2D-Mediated Immune RecognitionCD161 (human NKR-P1A) signaling in NK cells involves the activation of acid sphingomyelinaseInteraction between the human NK receptors and their ligands.The Impact of Glycosyl-Phosphatidyl-Inositol Anchored MICA Alleles on Novel NKG2D-Based Therapies.Analysis of the recovery of CD247 expression in a PID patient: Insights into the spontaneous repair of defective genes.Characterization of a human anti-tumoral NK cell population expanded after BCG treatment of leukocytes.Transfer of the human NKG2D ligands UL16 binding proteins (ULBP) 1-3 is related to lytic granule release and leads to ligand retransfer and killing of ULBP-recipient natural killer cells.Altered microRNA expression after infection with human cytomegalovirus leads to TIMP3 downregulation and increased shedding of metalloprotease substrates, including MICA.Cutting edge: the metalloproteinase ADAM17/TNF-alpha-converting enzyme regulates proteolytic shedding of the MHC class I-related chain B protein.Selective induction of expression of a ligand for the NKG2D receptor by proteasome inhibitors.A short half-life of ULBP1 at the cell surface due to internalization and proteosomal degradation.Intracellular trafficking of the HCMV immunoevasin UL16 depends on elements present in both its cytoplasmic and transmembrane domains.Genetic variability of the major histocompatibility complex class I homologue encoded by human cytomegalovirus leads to differential binding to the inhibitory receptor ILT2.Innate Immune Recognition of Double-stranded RNA Triggers Increased Expression of NKG2D Ligands After Virus Infection.The human NKG2D ligand ULBP2 can be expressed at the cell surface with or without a GPI anchor and both forms can activate NK cells.Impaired NK cell recognition of vemurafenib-treated melanoma cells is overcome by simultaneous application of histone deacetylase inhibitors.New Human Combined Immunodeficiency Due to IRF4 Deficiency Inherited by Uniparental Isodisomy.A GPI anchor explains the unique biological features of the common NKG2D-ligand allele MICA*008.NKG2D-ligands: putting everything under the same umbrella can be misleading.NKG2H-Expressing T Cells Negatively Regulate Immune Responses.Immunoassays for scarce tumour-antigens in exosomes: detection of the human NKG2D-Ligand, MICA, in tetraspanin-containing nanovesicles from melanoma.High sensitivity detection of extracellular vesicles immune-captured from urine by conventional flow cytometryNatural killer cell hyporesponsiveness and impaired development in a CD247-deficient patientTetraspanin-decorated extracellular vesicle-mimetics as a novel adaptable reference materialExosome beads array for multiplexed phenotyping in cancerBCG Therapy of Bladder Cancer Stimulates a Prolonged Release of the Chemoattractant CXCL10 (IP10) in Patient Urine
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description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Mar Valés-Gómez
@ast
Mar Valés-Gómez
@en
Mar Valés-Gómez
@es
Mar Valés-Gómez
@nl
type
label
Mar Valés-Gómez
@ast
Mar Valés-Gómez
@en
Mar Valés-Gómez
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Mar Valés-Gómez
@nl
prefLabel
Mar Valés-Gómez
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Mar Valés-Gómez
@en
Mar Valés-Gómez
@es
Mar Valés-Gómez
@nl
P106
P31
P496
0000-0001-7424-3206