about
GSG1L suppresses AMPA receptor-mediated synaptic transmission and uniquely modulates AMPA receptor kinetics in hippocampal neuronsPICK1 interacts with ABP/GRIP to regulate AMPA receptor trafficking.Trafficking of α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor (AMPA) receptor subunit GluA2 from the endoplasmic reticulum is stimulated by a complex containing Ca2+/calmodulin-activated kinase II (CaMKII) and PICK1 protein and by relSynaptic targeting of AMPA receptors is regulated by a CaMKII site in the first intracellular loop of GluA1.Synaptic anchorage of AMPA receptors by cadherins through neural plakophilin-related arm protein AMPA receptor-binding protein complexes.Increased expression of alpha-synuclein reduces neurotransmitter release by inhibiting synaptic vesicle reclustering after endocytosis.The stoichiometry of AMPA receptors and TARPs varies by neuronal cell type.Posttranslational regulation of AMPA receptor trafficking and functionNeurolastin, a Dynamin Family GTPase, Regulates Excitatory Synapses and Spine Density.An NMDA Receptor-Dependent Mechanism Underlies Inhibitory Synapse Development.The cell-autonomous role of excitatory synaptic transmission in the regulation of neuronal structure and functionCasein kinase 2 phosphorylates GluA1 and regulates its surface expression.Genetic analysis of neuronal ionotropic glutamate receptor subunits.Regulation of GABAergic synapse development by postsynaptic membrane proteins.A Rare Variant Identified Within the GluN2B C-Terminus in a Patient with Autism Affects NMDA Receptor Surface Expression and Spine Density.GSG1L regulates the strength of AMPA receptor-mediated synaptic transmission but not AMPA receptor kinetics in hippocampal dentate granule neurons.Subunit composition of synaptic AMPA receptors revealed by a single-cell genetic approach.Genetic inhibition of neurotransmission reveals role of glutamatergic input to dopamine neurons in high-effort behavior.Ferric Chelate Reductase 1 Like Protein (FRRS1L) Associates with Dynein Vesicles and Regulates Glutamatergic Synaptic Transmission.Molecular Dissection of Neuroligin 2 and Slitrk3 Reveals an Essential Framework for GABAergic Synapse Development.Genetic deletion of NMDA receptors suppresses GABAergic synaptic transmission in two distinct types of central neurons.Development of fast neurotransmitter synapses: General principle and recent progress.Role for VGLUT2 in selective vulnerability of midbrain dopamine neurons.Mossy Cells Control Adult Neural Stem Cell Quiescence and Maintenance through a Dynamic Balance between Direct and Indirect PathwaysShisa7 is a GABAA receptor auxiliary subunit controlling benzodiazepine actions
P50
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P50
description
onderzoeker
@nl
researcher ORCID: 0000-0001-6668-8432
@en
name
Wei Lu
@ast
Wei Lu
@en
Wei Lu
@nl
type
label
Wei Lu
@ast
Wei Lu
@en
Wei Lu
@nl
prefLabel
Wei Lu
@ast
Wei Lu
@en
Wei Lu
@nl
P106
P31
P496
0000-0001-6668-8432