Biochemical and histological changes in the small intestine of mice with dextran sulfate sodium colitis
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The Dipeptidyl Peptidase Family, Prolyl Oligopeptidase, and Prolyl Carboxypeptidase in the Immune System and Inflammatory Disease, Including AtherosclerosisBifidobacterium breve attenuates murine dextran sodium sulfate-induced colitis and increases regulatory T cell responsesGlutamine supplementation attenuates expressions of adhesion molecules and chemokine receptors on T cells in a murine model of acute colitis.The BAG-1 isoform BAG-1M regulates keratin-associated Hsp70 chaperoning of aPKC in intestinal cells during activation of inflammatory signaling.Effect of surface chemistry on nanoparticle interaction with gastrointestinal mucus and distribution in the gastrointestinal tract following oral and rectal administration in the mouse.MicroRNA-146a constrains multiple parameters of intestinal immunity and increases susceptibility to DSS colitis.Local corticosterone production and angiotensin-I converting enzyme shedding in a mouse model of intestinal inflammationInnate immune signalling at the intestinal epithelium in homeostasis and disease.Histopathological and morphometric changes induced by a dextran sodium sulfate (DSS) model in broilers.Unravelling the immunological roles of dipeptidyl peptidase 4 (DPP4) activity and/or structure homologue (DASH) proteins.The anxiolytic effect of Bifidobacterium longum NCC3001 involves vagal pathways for gut-brain communication.Reactivation of Intestinal Inflammation Is Suppressed by Catestatin in a Murine Model of Colitis via M1 Macrophages and Not the Gut Microbiota.β-Caryophyllene attenuates dextran sulfate sodium-induced colitis in mice via modulation of gene expression associated mainly with colon inflammation.The Anti-Inflammatory Effect and Intestinal Barrier Protection of HU210 Differentially Depend on TLR4 Signaling in Dextran Sulfate Sodium-Induced Murine Colitis.The enhanced susceptibility of ADAM-17 hypomorphic mice to DSS-induced colitis is not ameliorated by loss of RIPK3, revealing an unexpected function of ADAM-17 in necroptosis.Glutamine modulates acute dextran sulphate sodium-induced changes in small-intestinal intraepithelial γδ-T-lymphocyte expression in mice.The enhanced susceptibility of ADAM-17 hypomorphic mice to DSS-induced colitis is not ameliorated by loss of RIPK3, revealing an unexpected function of ADAM-17 in necroptosis.
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P2860
Biochemical and histological changes in the small intestine of mice with dextran sulfate sodium colitis
description
im Dezember 2011 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published on 01 December 2011
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в грудні 2011
@uk
name
Biochemical and histological c ...... dextran sulfate sodium colitis
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Biochemical and histological c ...... dextran sulfate sodium colitis
@nl
type
label
Biochemical and histological c ...... dextran sulfate sodium colitis
@en
Biochemical and histological c ...... dextran sulfate sodium colitis
@nl
prefLabel
Biochemical and histological c ...... dextran sulfate sodium colitis
@en
Biochemical and histological c ...... dextran sulfate sodium colitis
@nl
P2860
P50
P356
P1476
Biochemical and histological c ...... dextran sulfate sodium colitis
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P2093
Gordon S Howarth
Ross N Butler
P2860
P304
P356
10.1002/JCP.22682
P407
P577
2011-12-01T00:00:00Z