about
Tumor protein 53-induced nuclear protein 1 expression is repressed by miR-155, and its restoration inhibits pancreatic tumor developmentMolecular and functional characterization of the stress-induced protein (SIP) gene and its two transcripts generated by alternative splicing. SIP induced by stress and promotes cell deathCloning of IP15, a pancreatitis-induced gene whose expression inhibits cell growthTAp73 regulates the spindle assembly checkpoint by modulating BubR1 activityIsoform-specific p73 knockout mice reveal a novel role for delta Np73 in the DNA damage response pathwayCollagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions.Cloning and expression of the rat vacuole membrane protein 1 (VMP1), a new gene activated in pancreas with acute pancreatitis, which promotes vacuole formation.P53-dependent expression of the stress-induced protein (SIP).Assignment of tumor protein p53 induced nuclear protein 1 (TP53INP1) gene to human chromosome band 8q22 by in situ hybridization.TP53INP1s and homeodomain-interacting protein kinase-2 (HIPK2) are partners in regulating p53 activity.p73: a multifunctional protein in neurobiologyp73 in Cancer.Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreatic adenocarcinomaIER3 supports KRASG12D-dependent pancreatic cancer development by sustaining ERK1/2 phosphorylation.Pharmacological targeting of the protein synthesis mTOR/4E-BP1 pathway in cancer-associated fibroblasts abrogates pancreatic tumour chemoresistance.TAp73 depletion accelerates aging through metabolic dysregulationStromal SLIT2 impacts on pancreatic cancer-associated neural remodelingPancreatic preneoplastic lesions plasma signatures and biomarkers based on proteome profiling of mouse models.TAp73 is required for macrophage-mediated innate immunity and the resolution of inflammatory responses.Strengthened glycolysis under hypoxia supports tumor symbiosis and hexosamine biosynthesis in pancreatic adenocarcinoma.TAp73 knockout shows genomic instability with infertility and tumor suppressor functions.Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness.TAp73 is required for spermatogenesis and the maintenance of male fertility.Hypoxia induced tumor metabolic switch contributes to pancreatic cancer aggressiveness.TAp73 loss favors Smad-independent TGF-β signaling that drives EMT in pancreatic ductal adenocarcinoma.Consequences of DJ-1 upregulation following p53 loss and cell transformation.PAP/REG3A favors perineural invasion in pancreatic adenocarcinoma and serves as a prognostic marker.The conditional expression of KRAS G12D in mouse pancreas induces disorganization of endocrine islets prior the onset of ductal pre-cancerous lesions.LIF drives neural remodeling in pancreatic cancer and offers a new candidate biomarker.Stromal protein βig-h3 reprogrammes tumour microenvironment in pancreatic cancerMinimal information for studies of extracellular vesicles 2018 (MISEV2018): a position statement of the International Society for Extracellular Vesicles and update of the MISEV2014 guidelinesExpression Profiling in Pancreas during the Acute Phase of Pancreatitis Using cDNA MicroarraysCadherin-1 and cadherin-3 cooperation determines the aggressiveness of pancreatic ductal adenocarcinomaDendritic cell-based vaccination: powerful resources of immature dendritic cells against pancreatic adenocarcinomaSchwann cells support oncogenic potential of pancreatic cancer cells through TGFβ signalingCrosstalk between TAp73 and TGF-β in fibroblast regulates iNOS expression and Nrf2-dependent gene transcriptionCorrection: Schwann cells support oncogenic potential of pancreatic cancer cells through TGFβ signaling
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Richard Tomasini
@ast
Richard Tomasini
@en
Richard Tomasini
@es
Richard Tomasini
@nl
type
label
Richard Tomasini
@ast
Richard Tomasini
@en
Richard Tomasini
@es
Richard Tomasini
@nl
prefLabel
Richard Tomasini
@ast
Richard Tomasini
@en
Richard Tomasini
@es
Richard Tomasini
@nl
P106
P21
P31
P496
0000-0003-0869-0811