Hepatitis B virus-persistent infection and innate immunity defect: Cell-related or virus-related?

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Hepatitis B virus-persistent infection and innate immunity defect: Cell-related or virus-related?

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wetenschappelijk artikel

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наукова стаття, опублікована у вересні 2018

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Hepatitis B virus-persistent i ...... Cell-related or virus-related?

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Hepatitis B virus-persistent i ...... Cell-related or virus-related?

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Hepatitis B virus-persistent i ...... Cell-related or virus-related?

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Hepatitis B virus-persistent i ...... Cell-related or virus-related?

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Hepatitis B virus-persistent i ...... Cell-related or virus-related?

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Hepatitis B virus-persistent i ...... Cell-related or virus-related?

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P1476

Hepatitis B virus-persistent i ...... Cell-related or virus-related?

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Guo-Zhong Gong

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Jian Tang

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Jing Ma

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Rong-Juan Dai

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Zhen-Yu Wu

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P2860

The hepatitis B e antigen (HBeAg) targets and suppresses activation of the toll-like receptor signaling pathway

Peroxisomes are signaling platforms for antiviral innate immunity

Identification and characterization of MAVS, a mitochondrial antiviral signaling protein that activates NF-kappaB and IRF 3

Molecular biology of hepatitis B virus infection

Exosomes mediate hepatitis B virus (HBV) transmission and NK-cell dysfunction.

Genomic analysis of the host response to hepatitis B virus infection.

Toll-like receptor signaling inhibits hepatitis B virus replication in vivo.

Hepatitis B virus lacks immune activating capacity, but actively inhibits plasmacytoid dendritic cell function.

Downregulation of interleukin-18-mediated cell signaling and interferon gamma expression by the hepatitis B virus e antigen.

STING and the innate immune response to nucleic acids in the cytosol.

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233-241

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scholarly article

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10.12998/WJCC.V6.I9.233

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English

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2018-09-06T00:00:00Z

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30211203

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6134278

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