SGLT2 inhibitor dapagliflozin limits podocyte damage in proteinuric nondiabetic nephropathy

about

SGLT2 inhibitor dapagliflozin limits podocyte damage in proteinuric nondiabetic nephropathy

Item

http://www.wikidata.org/entity/Q57170120

description

wetenschappelijk artikel

@nl

наукова стаття, опублікована в серпні 2018

@uk

name

SGLT2 inhibitor dapagliflozin ...... inuric nondiabetic nephropathy

@en

SGLT2 inhibitor dapagliflozin ...... inuric nondiabetic nephropathy

@nl

type

Item

label

SGLT2 inhibitor dapagliflozin ...... inuric nondiabetic nephropathy

@en

SGLT2 inhibitor dapagliflozin ...... inuric nondiabetic nephropathy

@nl

prefLabel

SGLT2 inhibitor dapagliflozin ...... inuric nondiabetic nephropathy

@en

SGLT2 inhibitor dapagliflozin ...... inuric nondiabetic nephropathy

@nl

P1476

SGLT2 inhibitor dapagliflozin ...... inuric nondiabetic nephropathy

@en

P2093

Carlamaria Zoja

http://www.w3.org/2001/XMLSchema#string

Cristina Zanchi

http://www.w3.org/2001/XMLSchema#string

Daniela Corna

http://www.w3.org/2001/XMLSchema#string

Domenico Cerullo

http://www.w3.org/2001/XMLSchema#string

Marina Morigi

http://www.w3.org/2001/XMLSchema#string

Monica Locatelli

http://www.w3.org/2001/XMLSchema#string

Paola Cassis

http://www.w3.org/2001/XMLSchema#string

Sebastian Villa

http://www.w3.org/2001/XMLSchema#string

Simona Buelli

http://www.w3.org/2001/XMLSchema#string

P2860

Molecular mechanisms of diabetic kidney disease

FSGS-associated alpha-actinin-4 (K256E) impairs cytoskeletal dynamics in podocytes

In response to protein load podocytes reorganize cytoskeleton and modulate endothelin-1 gene: implication for permselective dysfunction of chronic nephropathies

Fractalkine and CX3CR1 mediate leukocyte capture by endothelium in response to Shiga toxin.

Laminin α2-mediated focal adhesion kinase activation triggers Alport glomerular pathogenesis.

Pathophysiologic implications of reduced podocyte number in a rat model of progressive glomerular injury.

Long-term treatment with the sodium glucose cotransporter 2 inhibitor, dapagliflozin, ameliorates glucose homeostasis and diabetic nephropathy in db/db mice.

Sodium-Glucose Linked Cotransporter-2 Inhibition Does Not Attenuate Disease Progression in the Rat Remnant Kidney Model of Chronic Kidney Disease.

Glomerular hyperfiltration and renal disease progression in type 2 diabetes.

eNOS deficiency predisposes podocytes to injury in diabetes.

P31

scholarly article

P356

10.1172/JCI.INSIGHT.98720

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Giuseppe Remuzzi

Ariela Benigni

P577

2018-08-09T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

30089717

http://www.w3.org/2001/XMLSchema#string

P932

6129124

http://www.w3.org/2001/XMLSchema#string