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Resveratrol treatment reduces cardiac progenitor cell dysfunction and prevents morpho-functional ventricular remodeling in type-1 diabetic ratsTitanium dioxide nanoparticles promote arrhythmias via a direct interaction with rat cardiac tissueGrowth factor-induced mobilization of cardiac progenitor cells reduces the risk of arrhythmias, in a rat model of chronic myocardial infarction.DIFFERENCES IN IONIC CURRENTS BETWEEN CANINE MYOCARDIAL AND PURKINJE CELLS.Parenchymal and Stromal Cells Contribute to Pro-Inflammatory Myocardial Environment at Early Stages of Diabetes: Protective Role of Resveratrol.Bioaccumulation of resveratrol metabolites in myocardial tissue is dose-time dependent and related to cardiac hemodynamics in diabetic rats.The histone deacetylase inhibitor suberoylanilide hydroxamic acid reduces cardiac arrhythmias in dystrophic mice.Characterization of the slowly inactivating sodium current INa2 in canine cardiac single Purkinje cells.Preservation of ventricular performance at early stages of diabetic cardiomyopathy involves changes in myocyte size, number and intercellular coupling.HDAC Inhibition Improves the Sarcoendoplasmic Reticulum Ca2+-ATPase Activity in Cardiac Myocytes.Antiarrhythmic effect of growth factor-supplemented cardiac progenitor cells in chronic infarcted heart.Correlation of alpha-skeletal actin expression, ventricular fibrosis and heart function with the degree of pressure overload cardiac hypertrophy in rats.Trimethylamine-N-Oxide (TMAO)-Induced Impairment of Cardiomyocyte Function and the Protective Role of Urolithin B-Glucuronide.Enhanced engraftment and repairing ability of human adipose-derived stem cells, conveyed by pharmacologically active microcarriers continuously releasing HGF and IGF-1, in healing myocardial infarction in rats.α-Actin isoform distribution in normal and failing human heart: a morphological, morphometric, and biochemical studyThe Histone Deacetylase Inhibitor Suberoylanilide Hydroxamic Acid (SAHA) Restores Cardiomyocyte Contractility in a Rat Model of Early DiabetesVulnerability to ventricular arrhythmias [corrected] and heterogeneity of action potential duration in normal ratsEffects of the alpha2-adrenergic/DA2-dopaminergic agonist CHF-1024 in preventing ventricular arrhythmogenesis and myocyte electrical remodeling, in a rat model of pressure-overload cardiac hypertrophy
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description
researcher ORCID: 0000-0003-4799-2804
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wetenschapper
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name
Leonardo Bocchi
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Leonardo Bocchi
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Leonardo Bocchi
@es
Leonardo Bocchi
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type
label
Leonardo Bocchi
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Leonardo Bocchi
@en
Leonardo Bocchi
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Leonardo Bocchi
@nl
prefLabel
Leonardo Bocchi
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Leonardo Bocchi
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Leonardo Bocchi
@es
Leonardo Bocchi
@nl
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15764829900
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0000-0003-4799-2804