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The expression of neuropeptide-induced excessive grooming behavior in dopamine D1 and D2 receptor-deficient miceUrocortin-deficient mice show hearing impairment and increased anxiety-like behaviorThe corticotropin-releasing factor receptor-1 pathway mediates the negative affective states of opiate withdrawal.Disruption of the CRF(2) receptor pathway decreases the somatic expression of opiate withdrawal.Understanding corticotropin releasing factor neurobiology: contributions from mutant mice.Targeted mutations of the corticotropin-releasing factor system: effects on physiology and behavior.CRF2 Receptor Deficiency Eliminates the Long-Lasting Vulnerability of Motivational States Induced by Opiate WithdrawalCRF₂ receptor-deficiency reduces recognition memory deficits and vulnerability to stress induced by cocaine withdrawal.The CRF1 and the CRF2 receptor mediate recognition memory deficits and vulnerability induced by opiate withdrawal.Genetic differences in response to novelty and spatial memory using a two-trial recognition task in mice.Lack of anticonvulsant tolerance and benzodiazepine receptor down regulation with imidazenil in rats.The corticotrophin-releasing factor/urocortin system regulates white fat browning in mice through paracrine mechanisms.Mice deficient for both corticotropin-releasing factor receptor 1 (CRFR1) and CRFR2 have an impaired stress response and display sexually dichotomous anxiety-like behavior.Lack of reward and locomotor stimulation induced by heroin in mu-opioid receptor-deficient mice.Decreased motivation to eat in mu-opioid receptor-deficient mice.Nitrous oxide (N2O) prevents latent pain sensitization and long-term anxiety-like behavior in pain and opioid-experienced rats.The corticotropin-releasing factor receptor-2 mediates the motivational effect of opiate withdrawal.CRF1 receptor-deficiency increases cocaine reward.CRF1 receptor-deficiency induces anxiety-like vulnerability to cocaine.CRF2 receptor-deficiency eliminates social behaviour deficits and vulnerability induced by cocaine.Abecarnil enhances recovery from diazepam tolerance.Disruption of the CRF/CRF1 receptor stress system exacerbates the somatic signs of opiate withdrawal.Binge-like eating in mice.Gender- and morphine dose-linked expression of spontaneous somatic opiate withdrawal in mice.CRF2 receptor-deficiency eliminates opiate withdrawal distress without impairing stress coping.Mice deficient for corticotropin-releasing hormone receptor-2 display anxiety-like behaviour and are hypersensitive to stress.A new place conditioning paradigm to study tolerance to opiates in mice.Dissociation of locomotor activation and suppression of food intake induced by CRF in CRFR1-deficient miceLong-lasting pseudo-social aggressive behavior in opiate-withdrawn miceSex-linked roles of the CRF1 and the CRF2 receptor in social behavior
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description
onderzoeker
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հետազոտող
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A Contarino
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A Contarino
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A Contarino
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A Contarino
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A Contarino
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A Contarino
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A Contarino
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P106
P31
P496
0000-0002-7286-6941