about
n-butylidenephthalide protects against dopaminergic neuron degeneration and α-synuclein accumulation in Caenorhabditis elegans models of Parkinson's diseaseExercise in an electrotactic flow chamber ameliorates age-related degeneration in Caenorhabditis elegans.A Disease Model of Muscle Necrosis Caused by Aeromonas dhakensis Infection in Caenorhabditis elegans.Hypoxia and the hypoxic response pathway protect against pore-forming toxins in C. elegans.WWP-1 is a novel modulator of the DAF-2 insulin-like signaling network involved in pore-forming toxin cellular defenses in Caenorhabditis elegans.MicroRNA-18a is elevated in prostate cancer and promotes tumorigenesis through suppressing STK4 in vitro and in vivo.Virulence diversity among bacteremic Aeromonas isolates: ex vivo, animal, and clinical evidences.Immobilization of the nematode Caenorhabditis elegans with addressable light-induced heat knockdown (ALINK).HLH-30/TFEB-mediated autophagy functions in a cell-autonomous manner for epithelium intrinsic cellular defense against bacterial pore-forming toxin in C. elegans.Enterohaemorrhagic Escherichia coli O157:H7 Shiga-like toxin 1 is required for full pathogenicity and activation of the p38 mitogen-activated protein kinase pathway in Caenorhabditis elegans.SESN-1 is a positive regulator of lifespan in Caenorhabditis elegans.A multi-omic analysis reveals the role of fumarate in regulating the virulence of enterohemorrhagic Escherichia coli.Loss of DNase II function in the gonad is associated with a higher expression of antimicrobial genes in Caenorhabditis elegans.
P50
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Chang-Shi Chen
@ast
Chang-Shi Chen
@en
Chang-Shi Chen
@es
Chang-Shi Chen
@nl
type
label
Chang-Shi Chen
@ast
Chang-Shi Chen
@en
Chang-Shi Chen
@es
Chang-Shi Chen
@nl
prefLabel
Chang-Shi Chen
@ast
Chang-Shi Chen
@en
Chang-Shi Chen
@es
Chang-Shi Chen
@nl
P106
P31
P496
0000-0002-8201-3635