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Gene therapy augments the efficacy of hematopoietic cell transplantation and fully corrects mucopolysaccharidosis type I phenotype in the mouse model.Relationship between post-cardiac arrest myocardial oxidative stress and myocardial dysfunction in the rat.Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice.Diabetes-associated cardiac fibrosis: Cellular effectors, molecular mechanisms and therapeutic opportunities.Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis.Inflammation as a therapeutic target in myocardial infarction: learning from past failures to meet future challenges.Ibuprofen plus isosorbide dinitrate treatment in the mdx mice ameliorates dystrophic heart structure.Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium.Postresuscitation treatment with argon improves early neurological recovery in a porcine model of cardiac arrest.Ranolazine ameliorates postresuscitation electrical instability and myocardial dysfunction and improves survival with good neurologic recovery in a rat model of cardiac arrest.Duration of Untreated Cardiac Arrest and Clinical Relevance of Animal Experiments: The Relationship Between the "No-Flow" Duration and the Severity of Post-Cardiac Arrest Syndrome in a Porcine Model.Left atrial remodeling, hypertrophy, and fibrosis in mouse models of heart failure.A novel echocardiographic method closely agrees with cardiac magnetic resonance in the assessment of left ventricular function in infarcted miceEarly kynurenine pathway activation following cardiac arrest in rats, pigs, and humansRanolazine prevents INaL enhancement and blunts myocardial remodelling in a model of pulmonary hypertensionAuthors' ReplyCharacterization of a mouse model of obesity-related fibrotic cardiomyopathy that recapitulates features of human heart failure with preserved ejection fractionMonocrotaline-induced pulmonary arterial hypertension: Time-course of injury and comparative evaluation of macitentan and Y-27632, a Rho kinase inhibitorProtective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving ProgramPrimary pulmonary arterial hypertension: Protocol to assess comprehensively in the rat the response to pharmacologic treatments
P50
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P50
description
researcher ORCID: 0000-0001-6432-3798
@en
wetenschapper
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name
Ilaria Russo
@ast
Ilaria Russo
@en
Ilaria Russo
@es
Ilaria Russo
@nl
type
label
Ilaria Russo
@ast
Ilaria Russo
@en
Ilaria Russo
@es
Ilaria Russo
@nl
prefLabel
Ilaria Russo
@ast
Ilaria Russo
@en
Ilaria Russo
@es
Ilaria Russo
@nl
P108
P106
P108
P1153
57196194874
P31
P496
0000-0001-6432-3798