Transcriptional Signature of Epidermal Keratinocytes Subjected toin VitroScratch Wounding Reveals Selective Roles for ERK1/2, p38, and Phosphatidylinositol 3-Kinase Signaling Pathways
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DrosophilaEmbryos as a Model for Wound-Induced Transcriptional Dynamics: Genetic Strategies to Achieve a Localized Wound ResponseThe DAF-16/FOXO transcription factor functions as a regulator of epidermal innate immunityCutaneous wound healing through paradoxical MAPK activation by BRAF inhibitors.ZFP36L1 is regulated by growth factors and cytokines in keratinocytes and influences their VEGF productionERK and phosphoinositide 3-kinase temporally coordinate different modes of actin-based motility during embryonic wound healing.Exploring hypotheses of the actions of TGF-beta1 in epidermal wound healing using a 3D computational multiscale model of the human epidermisThe basic helix-loop-helix/leucine zipper transcription factor USF2 integrates serum-induced PAI-1 expression and keratinocyte growthHypoxia regulates CD9-mediated keratinocyte migration via the P38/MAPK pathwayDelayed cutaneous wound healing and aberrant expression of hair follicle stem cell markers in mice selectively lacking Ctip2 in epidermis.Fibroin and sericin from Bombyx mori silk stimulate cell migration through upregulation and phosphorylation of c-Jun.Identification of a transcriptional signature for the wound healing continuumSerine proteolytic pathway activation reveals an expanded ensemble of wound response genes in Drosophila.SERPINE1 (PAI-1) is deposited into keratinocyte migration "trails" and required for optimal monolayer wound repairInhibition of inflammatory and proliferative responses of human keratinocytes exposed to the sesquiterpene lactones dehydrocostuslactone and costunolide.Amniotic Membrane Modifies the Genetic Program Induced by TGFß, Stimulating Keratinocyte Proliferation and Migration in Chronic Wounds.mTOR Activation by PI3K/Akt and ERK Signaling in Short ELF-EMF Exposed Human Keratinocytes.Oleanolic acid induces migration in Mv1Lu and MDA-MB-231 epithelial cells involving EGF receptor and MAP kinases activation.Loss of CAR promotes migration and proliferation of HaCaT cells, and accelerates wound healing in rats via Src-p38 MAPK pathwayInactivation of epidermal growth factor by Porphyromonas gingivalis as a potential mechanism for periodontal tissue damage.Comparative transcriptomic profiling of hydrogen peroxide signaling networks in zebrafish and human keratinocytes: Implications toward conservation, migration and wound healingInflammatory levels of nitric oxide inhibit airway epithelial cell migration by inhibition of the kinase ERK1/2 and activation of hypoxia-inducible factor-1 alpha.Multiple transcription factor codes activate epidermal wound-response genes in Drosophila.Wound repair at a glance.Src is activated by the nuclear receptor peroxisome proliferator-activated receptor β/δ in ultraviolet radiation-induced skin cancer.A review of transcriptomics in cutaneous chemical exposure.Mechanosignaling pathways in cutaneous scarring.Bullous pemphigoid: role of complement and mechanisms for blister formation within the lamina lucida.TLR3 drives IRF6-dependent IL-23p19 expression and p19/EBI3 heterodimer formation in keratinocytes.Zinc released from injured cells is acting via the Zn2+-sensing receptor, ZnR, to trigger signaling leading to epithelial repairFactor Xa stimulates proinflammatory and profibrotic responses in fibroblasts via protease-activated receptor-2 activation.Targeted Inhibition of PAI-1 Activity Impairs Epithelial Migration and Wound Closure Following Cutaneous Injury.PAI-1: An Integrator of Cell Signaling and Migration.Adrenergic signaling in human oral keratinocytes and wound repair.SERPINE1: A Molecular Switch in the Proliferation-Migration Dichotomy in Wound-"Activated" KeratinocytesIdentification of PCDH1 as a novel susceptibility gene for bronchial hyperresponsiveness.Heparin-binding EGF-like factor augments esophageal epithelial cell proliferation, migration and inhibits TRAIL-mediated apoptosis via EGFR/MAPK signaling.Protocadherin-1 polymorphisms are associated with eczema in two Dutch birth cohorts.Acute vascular endothelial growth factor expression during hypertrophy is muscle phenotype specific and localizes as a striated pattern within fibres.Effects of HB-EGF and epiregulin on wound healing of gingival cells in vitro.Activation of the human keratinocyte B1 bradykinin receptor induces expression and secretion of metalloproteases 2 and 9 by transactivation of epidermal growth factor receptor.
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Transcriptional Signature of Epidermal Keratinocytes Subjected toin VitroScratch Wounding Reveals Selective Roles for ERK1/2, p38, and Phosphatidylinositol 3-Kinase Signaling Pathways
description
article publié dans la revue scientifique Journal of Biological Chemistry
@fr
im März 2007 veröffentlichter wissenschaftlicher Artikel
@de
scientific article published in Journal of Biological Chemistry
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в березні 2007
@uk
name
Transcriptional Signature of E ...... ol 3-Kinase Signaling Pathways
@en
Transcriptional Signature of E ...... ol 3-Kinase Signaling Pathways
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type
label
Transcriptional Signature of E ...... ol 3-Kinase Signaling Pathways
@en
Transcriptional Signature of E ...... ol 3-Kinase Signaling Pathways
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prefLabel
Transcriptional Signature of E ...... ol 3-Kinase Signaling Pathways
@en
Transcriptional Signature of E ...... ol 3-Kinase Signaling Pathways
@nl
P2093
P2860
P50
P356
P1476
Transcriptional Signature of E ...... ol 3-Kinase Signaling Pathways
@en
P2093
Anne-Amandine Chassot
Chimène Moreilhon
Giorgos Fitsialos
Kevin LeBrigand
Laurent Turchi
Manal A. Dayem
Roser Buscà
P2860
P304
15090-15102
P356
10.1074/JBC.M606094200
P407
P577
2007-03-14T00:00:00Z