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Cutting Edge: BAFF Promotes Autoantibody Production via TACI-Dependent Activation of Transitional B Cells.The Autoimmune Risk Variant PTPN22 C1858T Alters B Cell Tolerance at Discrete Checkpoints and Differentially Shapes the Naive Repertoire.Cutting Edge: BAFF Overexpression Reduces Atherosclerosis via TACI-Dependent B Cell Activation.B cell-derived IL-6 initiates spontaneous germinal center formation during systemic autoimmunity.Integrated B Cell, Toll-like, and BAFF Receptor Signals Promote Autoantibody Production by Transitional B CellsFunctional Characterization of CD11c+ Age-Associated B Cells as Memory B CellsActivated CARD11 accelerates germinal center kinetics, promoting mTORC1 and terminal differentiationEfficient CRISPR/Cas9 Disruption of Autoimmune-Associated Genes Reveals Key Signaling Programs in Primary Human T CellsActivated PIK3CD drives innate B cell expansion yet limits B cell-intrinsic immune responsesTherapeutically relevant engraftment of a CRISPR-Cas9-edited HSC-enriched population with HbF reactivation in nonhuman primates
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
David J Rawlings
@ast
David J Rawlings
@en
David J Rawlings
@es
David J Rawlings
@nl
type
label
David J Rawlings
@ast
David J Rawlings
@en
David J Rawlings
@es
David J Rawlings
@nl
prefLabel
David J Rawlings
@ast
David J Rawlings
@en
David J Rawlings
@es
David J Rawlings
@nl
P106
P21
P31
P496
0000-0002-0810-1776