about
Altered BCR and TLR signals promote enhanced positive selection of autoreactive transitional B cells in Wiskott-Aldrich syndrome.B-cell intrinsic TLR7 signals promote depletion of the marginal zone in a murine model of Wiskott-Aldrich syndrome.B cell IFN-γ receptor signaling promotes autoimmune germinal centers via cell-intrinsic induction of BCL-6.Cutting Edge: BAFF Promotes Autoantibody Production via TACI-Dependent Activation of Transitional B Cells.Homology-Directed Recombination for Enhanced Engineering of Chimeric Antigen Receptor T Cells.Cutting Edge: BAFF Overexpression Reduces Atherosclerosis via TACI-Dependent B Cell Activation.B cell-derived IL-6 initiates spontaneous germinal center formation during systemic autoimmunity.TACI deletion protects against progressive murine lupus nephritis induced by BAFF overexpression
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Q36080732-3FACB74F-E638-44D8-A55D-BE60EE6BB1C4Q36371940-B8438D28-5EF1-4C47-BD2C-93EE8116C5F7Q36863150-A7473B09-8099-4872-836B-56C3FB14288DQ36905439-8917B0DA-4888-48D6-B8F6-FEC22DD3D0F4Q40277804-1D303ABE-818B-4453-982B-57413DE0A20DQ46774800-0F4CFE5B-1806-4434-929D-26F7A25CD805Q47815011-348C1CEB-24A5-451D-8846-F0EA4FBA56F4Q89119530-7280FDC4-B86A-4212-9234-F0EB7A3228EA
P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Holly M Jacobs
@ast
Holly M Jacobs
@en
Holly M Jacobs
@es
Holly M Jacobs
@nl
type
label
Holly M Jacobs
@ast
Holly M Jacobs
@en
Holly M Jacobs
@es
Holly M Jacobs
@nl
prefLabel
Holly M Jacobs
@ast
Holly M Jacobs
@en
Holly M Jacobs
@es
Holly M Jacobs
@nl
P106
P31
P496
0000-0003-3250-8258