about
Methodological aspects of the molecular and histological study of prostate cancer: focus on PTENStratification and therapeutic potential of PML in metastatic breast cancerPTEN mediates Notch-dependent stalk cell arrest in angiogenesisThe metabolic co-regulator PGC1α suppresses prostate cancer metastasis.HEY1 functions are regulated by its phosphorylation at Ser-68.Transcriptomic profiling of urine extracellular vesicles reveals alterations of CDH3 in prostate cancer.Pharmacological inhibition of fatty-acid oxidation synergistically enhances the effect of l-asparaginase in childhood ALL cells.mTORC1-dependent AMD1 regulation sustains polyamine metabolism in prostate cancer.Oil for the cancer engine: The cross-talk between oncogenic signaling and polyamine metabolism.PPARδ Elicits Ligand-Independent Repression of Trefoil Factor Family to Limit Prostate Cancer Growth.Low-dose statin treatment increases prostate cancer aggressiveness.Corrigendum: mTORC1-dependent AMD1 regulation sustains polyamine metabolism in prostate cancer.Erratum: The metabolic co-regulator PGC1α suppresses prostate cancer metastasisTargeting PML in triple negative breast cancer elicits growth suppression and senescence
P50
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P50
description
researcher ORCID id 0000-0002-1636-3944
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wetenschapper
@nl
name
Amaia Arruabarrena-Aristorena
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Amaia Arruabarrena-Aristorena
@en
Amaia Arruabarrena-Aristorena
@es
Amaia Arruabarrena-Aristorena
@nl
type
label
Amaia Arruabarrena-Aristorena
@ast
Amaia Arruabarrena-Aristorena
@en
Amaia Arruabarrena-Aristorena
@es
Amaia Arruabarrena-Aristorena
@nl
prefLabel
Amaia Arruabarrena-Aristorena
@ast
Amaia Arruabarrena-Aristorena
@en
Amaia Arruabarrena-Aristorena
@es
Amaia Arruabarrena-Aristorena
@nl
P106
P21
P31
P496
0000-0002-1636-3944