about
Gene expression of cyclooxygenase-1 and Ca(2+)-independent phospholipase A(2) is altered in rat hippocampus during normal agingSuppression of IGF-I signals in neural stem cells enhances neurogenesis and olfactory function during aging.Inhibition of NADPH oxidase promotes alternative and anti-inflammatory microglial activation during neuroinflammationThe Alzheimer's disease transcriptome mimics the neuroprotective signature of IGF-1 receptor-deficient neurons.Cyclooxygenase-1 inhibition reduces amyloid pathology and improves memory deficits in a mouse model of Alzheimer's disease.Insulin-like growth factor 1 receptor regulates hypothermia during calorie restriction.Regulatory T cells delay disease progression in Alzheimer-like pathology.IGF-1R Reduction Triggers Neuroprotective Signaling Pathways in Spinal Muscular Atrophy Mice.Blocking IGF Signaling in Adult Neurons Alleviates Alzheimer's Disease Pathology through Amyloid-β Clearance.Effect of a diet-induced n-3 PUFA depletion on cholinergic parameters in the rat hippocampus.Cerebral asymmetry and behavioral lateralization in rats chronically lacking n-3 polyunsaturated fatty acids.Disrupting IGF Signaling in Adult Mice Conditions Leanness, Resilient Energy Metabolism, and High Growth Hormone Pulses.Deleting IGF-1 receptor from forebrain neurons confers neuroprotection during stroke and upregulates endocrine somatotropin.
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description
researcher ORCID id 0000-0002-1427-581X
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wetenschapper
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name
Saba AÏD
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Saba AÏD
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Saba AÏD
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type
label
Saba AÏD
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Saba AÏD
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Saba AÏD
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prefLabel
Saba AÏD
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Saba AÏD
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Saba AÏD
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P106
P31
P496
0000-0002-1427-581X