about
Alzheimer's disease-like APP processing in wild-type mice identifies synaptic defects as initial steps of disease progression.A critical role for the glial-derived neuromodulator D-serine in the age-related deficits of cellular mechanisms of learning and memoryDecrease in calbindin content significantly alters LTP but not NMDA receptor and calcium channel propertiesImmunolesion of the cholinergic basal forebrain: effects on functional properties of hippocampal and septal neurons.Cholesterol 24-hydroxylase defect is implicated in memory impairments associated with Alzheimer-like Tau pathology.Circuit-specific changes in D-serine-dependent activation of the N-methyl-D-aspartate receptor in the aging hippocampus.The Chemokine MIP-1α/CCL3 impairs mouse hippocampal synaptic transmission, plasticity and memory.Unveiling an exceptional zymogen: the single-chain form of tPA is a selective activator of NMDA receptor-dependent signaling and neurotoxicitySomatostatinergic systems: an update on brain functions in normal and pathological aging.Deficits in memory and hippocampal long-term potentiation in mice with reduced calbindin D28K expression.GABAergic Microcircuits in Alzheimer's Disease Models.Contribution of the d-Serine-Dependent Pathway to the Cellular Mechanisms Underlying Cognitive AgingDifferent phosphatase-dependent mechanisms mediate long-term depression and depotentiation of long-term potentiation in mouse hippocampal CA1 area.Rescue of cognitive aging by long-lasting environmental enrichment exposure initiated before median lifespan.Decreased rhythmic GABAergic septal activity and memory-associated theta oscillations after hippocampal amyloid-beta pathology in the rat.Protein phosphatase 1-dependent transcriptional programs for long-term memory and plasticity.Reduction in glutamate uptake is associated with extrasynaptic NMDA and metabotropic glutamate receptor activation at the hippocampal CA1 synapse of aged rats.Rhythmical bursting activity and GABAergic mechanisms in the medial septum of normal and pertussis toxin-pretreated rats.Stimulation of phosphatidylinositol (PI) turnover may mediate the muscarinic suppression of the M-current in hippocampal pyramidal cells.Omega-3 fatty acids deficiency aggravates glutamatergic synapse and astroglial aging in the rat hippocampal CA1.A physiological role for GABAB receptors in the central nervous system.Pharmacological characterization of muscarinic responses in rat hippocampal pyramidal cells.The magnitude of hippocampal long term depression depends on the synaptic location of activated NR2-containing N-methyl-D-aspartate receptors.Preserved memory capacities in aged Lou/C/Jall rats.A role for the protein phosphatase 2B in altered hippocampal synaptic plasticity in the aged rat.βAPP Processing Drives Gradual Tau Pathology in an Age-Dependent Amyloid Rat Model of Alzheimer's Disease.Multiple cell types distinguished by physiological, pharmacological, and anatomic properties in nucleus HVc of the adult zebra finch.Somatosensory cortical neurons with an identifiable electrophysiological signature.Glutamatergic synaptic responses and long-term potentiation are impaired in the CA1 hippocampal area of calbindin D(28k)-deficient mice.Different effects of omega-conotoxin GVIA at excitatory and inhibitory synapses in rat CA1 hippocampal neurons.Presynaptic inhibitory effect of baclofen on hippocampal inhibitory synaptic transmission involves a pertussis toxin-sensitive G-protein.Age-related alterations in the properties of hippocampal pyramidal neurons among rat strains.Firing properties of anatomically identified neurons in the medial septum of anesthetized and unanesthetized restrained rats.Reduced serine racemase expression contributes to age-related deficits in hippocampal cognitive function.Selective impairment of some forms of synaptic plasticity by oligomeric amyloid-β peptide in the mouse hippocampus: implication of extrasynaptic NMDA receptors.Partial inhibition of PP1 alters bidirectional synaptic plasticity in the hippocampus.Age-related alterations of GABAergic input to CA1 pyramidal neurons and its control by nicotinic acetylcholine receptors in rat hippocampus.Motherhood-induced memory improvement persists across lifespan in rats but is abolished by a gestational stress.Reversal of age-related oxidative stress prevents hippocampal synaptic plasticity deficits by protecting D-serine-dependent NMDA receptor activation.A new neuronal target for β-amyloid peptide in the rat hippocampus.
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-8170-1196
@en
name
Patrick Dutar
@ast
Patrick Dutar
@en
Patrick Dutar
@es
Patrick Dutar
@nl
type
label
Patrick Dutar
@ast
Patrick Dutar
@en
Patrick Dutar
@es
Patrick Dutar
@nl
prefLabel
Patrick Dutar
@ast
Patrick Dutar
@en
Patrick Dutar
@es
Patrick Dutar
@nl
P106
P31
P496
0000-0002-8170-1196