about
Sialic acid-modified antigens impose tolerance via inhibition of T-cell proliferation and de novo induction of regulatory T cells.Antigen targeting reveals splenic CD169+ macrophages as promoters of germinal center B-cell responses.Functional CD169 on Macrophages Mediates Interaction with Dendritic Cells for CD8+ T Cell Cross-Priming.Dendritic Cell Migration to Skin-Draining Lymph Nodes Is Controlled by Dermatan Sulfate and Determines Adaptive Immunity Magnitude.The identification and developmental requirements of colonic CD169⁺ macrophages.The inhibitory CD200R is differentially expressed on human and mouse T and B lymphocytes.Ligation of CD200R by CD200 is not required for normal murine myelopoiesis
P50
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P50
description
researcher ORCID ID = 0000-0001-8731-0615
@en
wetenschapper
@nl
name
Henrike Veninga
@ast
Henrike Veninga
@en
Henrike Veninga
@es
Henrike Veninga
@nl
type
label
Henrike Veninga
@ast
Henrike Veninga
@en
Henrike Veninga
@es
Henrike Veninga
@nl
prefLabel
Henrike Veninga
@ast
Henrike Veninga
@en
Henrike Veninga
@es
Henrike Veninga
@nl
P106
P1153
22942809800
P21
P31
P496
0000-0001-8731-0615