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Hypoxia-induced phrenic long-term facilitation: emergent propertiesAdrenergic α₁ receptor activation is sufficient, but not necessary for phrenic long-term facilitation.P2Y1 receptor-mediated potentiation of inspiratory motor output in neonatal rat in vitro.Intermittent Hypoxia-Induced Spinal Inflammation Impairs Respiratory Motor Plasticity by a Spinal p38 MAP Kinase-Dependent MechanismPhrenic long-term facilitation requires PKCθ activity within phrenic motor neurons.ATP in central respiratory control: a three-part signaling system.Isolated in vitro brainstem-spinal cord preparations remain important tools in respiratory neurobiology.The impact of inflammation on respiratory plasticity.Interpreting deactivations in neuroimaging.Glia contribute to the purinergic modulation of inspiratory rhythm-generating networks.Tripartite purinergic modulation of central respiratory networks during perinatal development: the influence of ATP, ectonucleotidases, and ATP metabolites.Cyclooxygenase enzyme activity does not impair respiratory motor plasticity after one night of intermittent hypoxia.Gestational intermittent hypoxia increases susceptibility to neuroinflammation and alters respiratory motor control in neonatal rats.Substance P modulation of hypoglossal motoneuron excitability during development: changing balance between conductances.P2Y1 receptor modulation of the pre-Bötzinger complex inspiratory rhythm generating network in vitro.IL-1 receptor activation undermines respiratory motor plasticity after systemic inflammationSpinal protein phosphatase 1 constrains respiratory plasticity after sustained hypoxiaOne bout of neonatal inflammation impairs adult respiratory motor plasticity in male and female rats
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P50
description
Forscher
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chercheur
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investigador
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onderzoeker
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researcher
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հետազոտող
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研究者
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name
Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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Adrianne Huxtable
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